Literature DB >> 31439680

Intragraft donor-specific anti-HLA antibodies in phenotypes of chronic lung allograft dysfunction.

Annelore Sacreas1, Jean-Luc Taupin2, Marie-Paule Emonds3,4, Liesbeth Daniëls3, Dirk E Van Raemdonck1,5, Robin Vos1, Geert M Verleden1, Bart M Vanaudenaerde1, Antoine Roux6, Stijn E Verleden7.   

Abstract

INTRODUCTION: Circulating anti-human leukocyte antigen (HLA) serum donor-specific antibodies (sDSAs) increase the risk of chronic lung allograft dysfunction (CLAD) and mortality. Discrepancies between serological and pathological/clinical findings are common. Therefore, we aimed to assess the presence of tissue-bound graft DSAs (gDSAs) in CLAD explant tissue compared with sDSAs.
METHODS: Tissue cores, obtained from explant lungs of unused donors (n=10) and patients with bronchiolitis obliterans syndrome (BOS; n=18) and restrictive allograft syndrome (RAS; n=18), were scanned with micro-computed tomography before elution of antibodies. Total IgG levels were measured via ELISA. Anti-HLA class I and II IgG gDSAs were identified using Luminex single antigen beads and compared with DSAs found in serum samples.
RESULTS: Overall, mean fluorescence intensity was higher in RAS eluates compared with BOS and controls (p<0.0001). In BOS, two patients were sDSA+/gDSA+ and two patients were sDSA-/gDSA+. In RAS, four patients were sDSA+/gDSA+, one patient was sDSA+/gDSA- and five patients were sDSA-/gDSA+. Serum and graft results combined, DSAs were more prevalent in RAS compared with BOS (56% versus 22%; p=0.04). There was spatial variability in gDSA detection in one BOS patient and three RAS patients, who were all sDSA-. Total graft IgG levels were higher in RAS than BOS (p<0.0001) and in gDSA+ versus gDSA- (p=0.0008), but not in sDSA+ versus sDSA- (p=0.33). In RAS, total IgG levels correlated with fibrosis (r= -0.39; p=0.02).
CONCLUSIONS: This study underlines the potential of gDSA assessment as complementary information to sDSA findings. The relevance and applications of gDSAs need further investigation.
Copyright ©ERS 2019.

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Year:  2019        PMID: 31439680     DOI: 10.1183/13993003.00847-2019

Source DB:  PubMed          Journal:  Eur Respir J        ISSN: 0903-1936            Impact factor:   16.671


  3 in total

1.  Immune Checkpoints Expression in Chronic Lung Allograft Rejection.

Authors:  Ilaria Righi; Valentina Vaira; Letizia Corinna Morlacchi; Giorgio Alberto Croci; Valeria Rossetti; Francesco Blasi; Stefano Ferrero; Mario Nosotti; Lorenzo Rosso; Mario Clerici
Journal:  Front Immunol       Date:  2021-08-13       Impact factor: 7.561

2.  Activation of Humoral Immunity during the Pathogenesis of Experimental Chronic Lung Allograft Dysfunction.

Authors:  Martin Reichert; Srebrena Atanasova; Kathrin Petri; Marian Kampschulte; Baktybek Kojonazarov; Gabriele Fuchs-Moll; Gabriele A Krombach; Winfried Padberg; Veronika Grau
Journal:  Int J Mol Sci       Date:  2022-07-23       Impact factor: 6.208

Review 3.  B Cell Immunity in Lung Transplant Rejection - Effector Mechanisms and Therapeutic Implications.

Authors:  Birte Ohm; Wolfgang Jungraithmayr
Journal:  Front Immunol       Date:  2022-03-07       Impact factor: 7.561

  3 in total

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