Literature DB >> 31381526

AVXS-101 (Onasemnogene Abeparvovec) for SMA1: Comparative Study with a Prospective Natural History Cohort.

Samiah A Al-Zaidy1,2, Stephen J Kolb3,4, Linda Lowes1,2, Lindsay N Alfano1,2, Richard Shell2, Kathleen R Church1, Sukumar Nagendran5, Douglas M Sproule5, Douglas E Feltner5, Courtney Wells5, Francis Ogrinc5, Melissa Menier5, James L'Italien5, W David Arnold3, John T Kissel2, Brian K Kaspar5, Jerry R Mendell1,2,3.   

Abstract

BACKGROUND: Spinal muscular atrophy type 1 (SMA1) is the leading genetic cause of infant mortality for which therapies, including AVXS-101 (onasemnogene abeparvovec, Zolgensma®) gene replacement therapy, are emerging.
OBJECTIVE: This study evaluated the effectiveness of AVXS-101 in infants with spinal muscular atrophy type 1 (SMA1) compared with a prospective natural history cohort and a cohort of healthy infants.
METHODS: Twelve SMA1 infants received the proposed therapeutic dose of AVXS-101 (NCT02122952). Where possible, the following outcomes were compared with a natural history cohort of SMA1 infants (n = 16) and healthy infants (n = 27) enrolled in the NeuroNEXT (NN101) study (NCT01736553): event-free survival, CHOP-INTEND scores, motor milestone achievements, compound muscle action potential (CMAP), and adverse events.
RESULTS: Baseline characteristics of SMA1 infants in the AVXS-101 and NN101 studies were similar in age and genetic profile. The proportion of AVXS-101-treated infants who survived by 24 months of follow-up was higher compared with the NN101 study (100% vs 38%, respectively). The average baseline CHOP-INTEND score for NN101 SMA1 infants was 20.3, worsening to 5.3 by age 24 months; the average baseline score in AVXS-101-treated infants was 28.2, improving to 56.5 by age 24 months. Infants receiving AVXS-101 achieved motor milestones, such as sitting unassisted and walking. Improvements in CMAP peak area were observed in AVXS-101-treated infants at 6 and 24 months (means of 1.1 and 3.2 mV/s, respectively).
CONCLUSIONS: In this study, AVXS-101 increased the probability of survival, rapidly improved motor function, and enabled motor milestone achievement in SMA1 infants.

Entities:  

Keywords:  Gene therapy; natural history; neuromuscular diseases; spinal muscular atrophy

Mesh:

Year:  2019        PMID: 31381526     DOI: 10.3233/JND-190403

Source DB:  PubMed          Journal:  J Neuromuscul Dis


  34 in total

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