Literature DB >> 31376376

Type B Aortic Dissection in Young Individuals With Confirmed and Presumed Heritable Thoracic Aortic Disease.

Sherene Shalhub1, Mary J Roman2, Kim A Eagle3, Scott A LeMaire4, Qianzi Zhang4, Artur Evangelista5, Dianna M Milewicz6.   

Abstract

BACKGROUND: To investigate clinical course of patients with type B aortic dissection (TBAD) occurring at a young age with confirmed or suspected heritable thoracic aortic disease.
METHODS: Individuals with TBAD occurring at an age <50 years enrolled in the National Registry of the Genetically Triggered Thoracic Aortic Aneurysms and Cardiovascular Conditions Consortium were selected for analysis. Three cohorts were compared: Marfan syndrome (MFS TBAD), nonsyndromic familial TBAD (FTBAD), and sporadic TBAD. Demographics, comorbidities, aortic dissection details, and repair were compared.
RESULTS: A total of 150 individuals met inclusion criteria (mean age at TBAD, 36.9 ± 9 years): 73 MFS TBAD, 42 FTBAD, and 35 sporadic TBAD. The cohort of sporadic TBAD had more male patients (71.4%) and fewer individuals of European descent (51.4%) compared with MFS TBAD (57.5% male, 84.9% European descent) and FTBAD (59.5% male, 90.5% European descent). There was a stepwise increase in hypertension prevalence across the cohorts (28.8% MFS, 59.5% FTBAD, 71.4% sporadic TBAD, P < .001). Repair of the descending thoracic aorta was performed in 92 cases (67.1% in MFS, 61.9% in FTBAD, and 48.6% sporadic TBAD, P = .18) at a mean of 3.4 ± 5.4 years from TBAD. The repair extent varied. The largest extent of repair was in MFS TBAD, in which thoracoabdominal aortic aneurysm repair was performed in 56.2% compared with 35.7% FTBAD and 17.1% sporadic TBAD (P < .001).
CONCLUSIONS: Control of hypertension is an essential component of care to decrease the risk of TBAD. Over half of the young individuals with TBAD require aortic repair, and individuals with MFS undergo a larger anatomical extent of repair after TBAD.
Copyright © 2020 The Society of Thoracic Surgeons. Published by Elsevier Inc. All rights reserved.

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Mesh:

Year:  2019        PMID: 31376376     DOI: 10.1016/j.athoracsur.2019.07.004

Source DB:  PubMed          Journal:  Ann Thorac Surg        ISSN: 0003-4975            Impact factor:   4.330


  4 in total

1.  Expanding ACTA2 genotypes with corresponding phenotypes overlapping with smooth muscle dysfunction syndrome.

Authors:  Anita Kaw; Kaveeta Kaw; Ellen M Hostetler; Ana Beleza-Meireles; Adam Smith-Collins; Catherine Armstrong; Ingrid Scurr; Timothy Cotts; Rajani Aatre; Michael J Bamshad; Dawn Earl; Abraham Groner; Katherine Agre; Yehuda Raveh; Callie S Kwartler; Dianna M Milewicz
Journal:  Am J Med Genet A       Date:  2022-05-14       Impact factor: 2.578

2.  Cardiovascular Outcomes in Aortopathy: GenTAC Registry of Genetically Triggered Aortic Aneurysms and Related Conditions.

Authors:  Kathryn W Holmes; Sheila Markwardt; Kim A Eagle; Richard B Devereux; Jonathan W Weinsaft; Federico M Asch; Scott A LeMaire; Cheryl L Maslen; Howard K Song; Dianna M Milewicz; Siddharth K Prakash; Dongchuan Guo; Shaine A Morris; Reed E Pyeritz; Rita C Milewski; William J Ravekes; H C Dietz; Ralph V Shohet; Michael Silberbach; Mary J Roman
Journal:  J Am Coll Cardiol       Date:  2022-05-31       Impact factor: 27.203

Review 3.  Marfan syndrome.

Authors:  Dianna M Milewicz; Alan C Braverman; Julie De Backer; Shaine A Morris; Catherine Boileau; Irene H Maumenee; Guillaume Jondeau; Arturo Evangelista; Reed E Pyeritz
Journal:  Nat Rev Dis Primers       Date:  2021-09-02       Impact factor: 65.038

4.  Association of genetic variants in ULK4 with the age of first onset of type B aortic dissection.

Authors:  Lihong Huang; Jiaqi Tang; Lijuan Lin; Ruihan Wang; Feng Chen; Yongyue Wei; Yi Si; Weiguo Fu
Journal:  Front Genet       Date:  2022-09-02       Impact factor: 4.772

  4 in total

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