Literature DB >> 31372663

Immunoexpression of canonical Wnt and NF-κB signaling pathways in the temporomandibular joint of arthritic rats.

Luane Macêdo de Sousa1, Joana Maria Dos Santos Alves2, Conceição da Silva Martins1, Karuza Maria Alves Pereira1,2, Paula Goes1,2, Delane Viana Gondim3,4.   

Abstract

OBJECTIVE: To investigate the participation of canonical Wnt and NF-κB signaling pathways in an experimental model of chronic arthritis induced by methylated bovine serum albumin (mBSA) in rat temporomandibular joint (TMJ).
MATERIALS AND METHODS: Wistar rats were sensitized by mBSA+Complete Freund Adjuvant (CFA)/Incomplete Freund Adjuvant (IFA) on the first 14 days (1 ×/week). Subsequently, they received 1, 2 or 3 mBSA or saline solution injections into the TMJ (1 ×/week). Hypernociceptive threshold was assessed during the whole experimental period. 24 h after the mBSA injections, the TMJs were removed for histopathological and immunohistochemical analyses for TNF-α, IL-1β, NF-κB, RANKL, Wnt-10b, β-catenin and DKK1.
RESULTS: The nociceptive threshold was significantly reduced after mBSA injections. An inflammatory infiltrate and thickening of the synovial membrane were observed only after mBSA booster injections. Immunolabeling of TNF-α, IL-1β and Wnt-10b was increased in the synovial membrane in arthritic groups. The immunoexpression of nuclear β-catenin was significantly higher only in the group that received 2 booster TMJ injections. However, NF-κB, RANKL and DKK1 immunoexpression were increased only in animals with 3 mBSA intra-articular injections.
CONCLUSION: Our results suggest that canonical Wnt and NF-κB signaling pathways participate in the hypernociception and inflammatory response in TMJ synovial membrane during the development of rheumatoid arthritis in rats.

Entities:  

Keywords:  Arthritis; Inflammation; NF-kappa B kinase; Rheumatoid; Synovial membrane; Temporomandibular joint; Wnt signaling pathway

Mesh:

Substances:

Year:  2019        PMID: 31372663     DOI: 10.1007/s00011-019-01274-4

Source DB:  PubMed          Journal:  Inflamm Res        ISSN: 1023-3830            Impact factor:   4.575


  54 in total

1.  Differential expression of WNTs and FRPs in the synovium of rheumatoid arthritis and osteoarthritis.

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2.  Stimulation of bone resorption and inhibition of bone formation in vitro by human tumour necrosis factors.

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4.  Expression of β-catenin in rheumatoid arthritis fibroblast-like synoviocytes.

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8.  Interleukin 6/Wnt interactions in rheumatoid arthritis: interleukin 6 inhibits Wnt signaling in synovial fibroblasts and osteoblasts.

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Review 9.  Crosstalk between Wnt/β-Catenin and NF-κB Signaling Pathway during Inflammation.

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Journal:  PLoS One       Date:  2016-01-07       Impact factor: 3.240

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  7 in total

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Review 3.  The Effects of Crocin on Bone and Cartilage Diseases.

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5.  C-Reactive Protein Knockout Attenuates Temporomandibular Joint Inflammation in Rats.

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6.  Antinociceptive Actions of Botulinum Toxin A1 on Immunogenic Hypersensitivity in Temporomandibular Joint of Rats.

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7.  Up-regulated microRNA-411 or declined RIPK1 inhibits proliferation and promotes apoptosis of synoviocytes in rheumatoid arthritis mice via decreased NF-κB pathway.

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Journal:  Cell Cycle       Date:  2020-03-01       Impact factor: 4.534

  7 in total

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