Literature DB >> 31303531

PKM2 upregulation promotes malignancy and indicates poor prognosis for intrahepatic cholangiocarcinoma.

Ze Qian1, Wendi Hu1, Zhen Lv1, Hua Liu1, Diyu Chen1, Yacong Wang2, Jian Wu3, Shusen Zheng4.   

Abstract

BACKGROUND: Although pyruvate kinase M2 (PKM2) has been shown to be among the crucial enzymes that regulate aerobic glycolysis in multiple tumour cells, its role in the treatment and prognosis of intrahepatic cholangiocarcinoma (ICC) remains unclear. This study primarily aimed to determine whether the expression status of PKM2 is potentially associated with the clinical outcomes of ICC.
METHODS: PKM2 expression was evaluated in ICC cell lines and tissues via real-time quantitative reverse-transcription polymerase chain reaction, immunofluorescence assays, and Western blot, and its prognostic value was determined according to its impact on the overall survival of patients.
RESULTS: We found that PKM2 is highly expressed in ICC, and this was correlated with patient survival. Moreover, we found that PKM2 knockdown could considerably inhibit ICC cell proliferation, invasion, and migration in vitro.
CONCLUSIONS: PKM2 was overexpressed in ICC, and it may regulate proliferation, invasion, and migration and lead to poor prognosis. Thus, PKM2 might be a potential independent prognostic factor for ICC.
Copyright © 2019 Elsevier Masson SAS. All rights reserved.

Entities:  

Keywords:  Cell proliferation; Intrahepatic cholangiocarcinoma; Migration; Prognosis; Pyruvate kinase

Mesh:

Substances:

Year:  2019        PMID: 31303531     DOI: 10.1016/j.clinre.2019.06.008

Source DB:  PubMed          Journal:  Clin Res Hepatol Gastroenterol        ISSN: 2210-7401            Impact factor:   2.947


  3 in total

Review 1.  Impact of Alternative Splicing Variants on Liver Cancer Biology.

Authors:  Jose J G Marin; Maria Reviejo; Meraris Soto; Elisa Lozano; Maitane Asensio; Sara Ortiz-Rivero; Carmen Berasain; Matias A Avila; Elisa Herraez
Journal:  Cancers (Basel)       Date:  2021-12-21       Impact factor: 6.639

2.  Deoxyribonuclease 1-like 3 Inhibits Hepatocellular Carcinoma Progression by Inducing Apoptosis and Reprogramming Glucose Metabolism.

Authors:  Yusha Xiao; Kang Yang; Pengpeng Liu; Dong Ma; Ping Lei; Quanyan Liu
Journal:  Int J Biol Sci       Date:  2022-01-01       Impact factor: 6.580

3.  Metformin overcomes metabolic reprogramming-induced resistance of skin squamous cell carcinoma to photodynamic therapy.

Authors:  Marta Mascaraque-Checa; María Gallego-Rentero; Jimena Nicolás-Morala; Mikel Portillo-Esnaola; José M Cuezva; Salvador González; Yolanda Gilaberte; Ángeles Juarranz
Journal:  Mol Metab       Date:  2022-04-09       Impact factor: 8.568

  3 in total

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