Literature DB >> 31269827

The elevated CXCL5 levels in circulation are associated with lung function decline in COPD patients and cigarette smoking-induced mouse model of COPD.

Jun Chen1,2, Luqi Dai1,2, Tao Wang1,2, Junyun He3, Yashu Wang4, Fuqiang Wen1,2.   

Abstract

Introduction: C-X-C motif chemokine 5 is primarily chemotactic for neutrophils and previously shown to increase in the bronchoalveolar lavage fluid of patients with chronic obstructive pulmonary disease. However, whether C-X-C motif chemokine 5 levels correlate with lung function decline in patients or mouse model of chronic obstructive pulmonary disease was not clear.
Methods: The mouse model was induced by cigarette smoke exposure. Plasma/serum and bronchoalveolar lavage fluid were obtained from patients and mouse model of chronic obstructive pulmonary disease; C-X-C motif chemokine 5 levels were assessed and correlated with lung functions and granulocyte-colony stimulating factor levels, respectively.
Results: The C-X-C motif chemokine 5 levels increased and correlated to granulocyte-colony stimulating factor levels in both plasma/serum and bronchoalveolar lavage fluid obtained from patients and mouse model of chronic obstructive pulmonary disease. Circulating levels of C-X-C motif chemokine 5 correlated to lung functions decline in patients and mouse model. Conclusions: Granulocyte-colony stimulating factor might coordinate with C-X-C motif chemokine 5 in the pathogenesis of neutrophilic inflammation in chronic obstructive pulmonary disease. Circulating C-X-C motif chemokine 5 might serve as a potential blood-based biomarker to add additional modest predictive value on the preliminary screening and diagnosis of chronic obstructive pulmonary disease. Key messages Circulating C-X-C motif chemokine 5 might serve as a potential blood-based biomarker to add additional modest predictive value on the preliminary screening and diagnosis of COPD. Granulocyte-colony stimulating factor might coordinate with C-X-C motif chemokine 5 in the pathogenesis of neutrophilic inflammation in chronic obstructive pulmonary disease.

Entities:  

Keywords:  C-X-C motif chemokine 5; Chronic obstructive pulmonary disease; cigarette smoke; granulocyte-colony stimulating factor; lung function decline; mouse model of COPD; nose-only exposure; receiver operating characteristic

Mesh:

Substances:

Year:  2019        PMID: 31269827      PMCID: PMC7877878          DOI: 10.1080/07853890.2019.1639809

Source DB:  PubMed          Journal:  Ann Med        ISSN: 0785-3890            Impact factor:   4.709


  46 in total

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4.  NH2- and COOH-terminal truncations of murine granulocyte chemotactic protein-2 augment the in vitro and in vivo neutrophil chemotactic potency.

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8.  The Role of Mean Platelet Volume in Chronic Obstructive Pulmonary Disease Exacerbation.

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Authors:  Jun Chen; Linhong Deng; Daniela Dreymüller; Xuemei Jiang; Jiaoyue Long; Yiyuan Duan; Yue Wang; Mingzhi Luo; Feng Lin; Lizhen Mao; Bernd Müller; Garrit Koller; Jörg W Bartsch
Journal:  Sci Rep       Date:  2016-07-26       Impact factor: 4.379

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2.  Complement factor H deficiency combined with smoking promotes retinal degeneration in a novel mouse model.

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3.  p300/Sp1-Mediated High Expression of p16 Promotes Endothelial Progenitor Cell Senescence Leading to the Occurrence of Chronic Obstructive Pulmonary Disease.

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Journal:  Mediators Inflamm       Date:  2021-08-19       Impact factor: 4.711

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5.  Epithelial Ablation of Miro1/Rhot1 GTPase Augments Lung Inflammation by Cigarette Smoke.

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  5 in total

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