Literature DB >> 31194225

Open-label Clinical Trial of Niraparib Combined With Pembrolizumab for Treatment of Advanced or Metastatic Triple-Negative Breast Cancer.

Shaveta Vinayak1,2, Sara M Tolaney3, Lee Schwartzberg4, Monica Mita5, Georgia McCann6, Antoinette R Tan7, Andrea E Wahner-Hendrickson8, Andres Forero9, Carey Anders10,11, Gerburg M Wulf12, Patrick Dillon13, Filipa Lynce14, Corrine Zarwan15, John K Erban16, Yinghui Zhou17, Nathan Buerstatte17, Julie R Graham17, Sujata Arora17, Bruce J Dezube17, Melinda L Telli18.   

Abstract

IMPORTANCE: Poly(adenosine diphosphate-ribose) polymerase inhibitor and anti-programmed death receptor-1 inhibitor monotherapy have shown limited clinical activity in patients with advanced triple-negative breast cancer (TNBC).
OBJECTIVE: To evaluate the clinical activity (primary) and safety (secondary) of combination treatment with niraparib and pembrolizumab in patients with advanced or metastatic TNBC. DESIGN, SETTING, AND PARTICIPANTS: This open-label, single-arm, phase 2 study enrolled 55 eligible patients with advanced or metastatic TNBC irrespective of BRCA mutation status or programmed death-ligand 1 (PD-L1) expression at 34 US sites. Data were collected from January 3, 2017, through October 29, 2018, and analyzed from October 29, 2018, through February 27, 2019.
INTERVENTIONS: Patients were administered 200 mg of oral niraparib once daily in combination with 200 mg of intravenous pembrolizumab on day 1 of each 21-day cycle. MAIN OUTCOMES AND MEASURES: The primary end point was objective response rate (ORR) per the Response Evaluation Criteria in Solid Tumors, version 1.1. Secondary end points were safety, disease control rate (DCR; complete response plus partial response plus stable disease), duration of response (DOR), progression-free survival (PFS), and overall survival.
RESULTS: Within the full study population of 55 women (median age, 54 years [range, 32-90 years]), 5 patients had confirmed complete responses, 5 had confirmed partial responses, 13 had stable disease, and 24 had progressive disease. In the efficacy-evaluable population (n = 47), ORR included 10 patients (21%; 90% CI, 12%-33%) and DCR included 23 (49%; 90% CI, 36%-62%). Median DOR was not reached at the time of the data cutoff, with 7 patients still receiving treatment at the time of analysis. In 15 evaluable patients with tumor BRCA mutations, ORR included 7 patients(47%; 90% CI, 24%-70%), DCR included 12 (80%; 90% CI, 56%-94%), and median PFS was 8.3 months (95% CI, 2.1 months to not estimable). In 27 evaluable patients with BRCA wild-type tumors, ORR included 3 patients (11%; 90% CI, 3%-26%), DCR included 9 (33%; 90% CI, 19%-51%), and median PFS was 2.1 months (95% CI, 1.4-2.5 months). The most common treatment-related adverse events of grade 3 or higher were anemia (10 [18%]), thrombocytopenia (8 [15%]), and fatigue (4 [7%]). Immune-related adverse events were reported in 8 patients (15%) and were grade 3 in 2 patients (4%); no new safety signals were detected. CONCLUSIONS AND RELEVANCE: Combination niraparib plus pembrolizumab provides promising antitumor activity in patients with advanced or metastatic TNBC, with numerically higher response rates in those with tumor BRCA mutations. The combination therapy was safe with a tolerable safety profile, warranting further investigation. TRIAL REGISTRATION: ClinicalTrials.gov identifier: NCT02657889.

Entities:  

Year:  2019        PMID: 31194225      PMCID: PMC6567845          DOI: 10.1001/jamaoncol.2019.1029

Source DB:  PubMed          Journal:  JAMA Oncol        ISSN: 2374-2437            Impact factor:   33.006


  85 in total

1.  Genomic Methods Identify Homologous Recombination Deficiency in Pancreas Adenocarcinoma and Optimize Treatment Selection.

Authors:  Wungki Park; Jiapeng Chen; Nadeem Riaz; Eileen M O'Reilly; Joanne F Chou; Anna M Varghese; Kenneth H Yu; Winston Wong; Marinela Capanu; Vinod Balachandran; Caitlin A McIntyre; Imane El Dika; Danny N Khalil; James J Harding; Nima Ghalehsari; Zoe McKinnell; Sree B Chalasani; Vladimir Makarov; Pier Selenica; Xin Pei; Nicolas Lecomte; David P Kelsen; Ghassan K Abou-Alfa; Mark E Robson; Liying Zhang; Michael F Berger; Nikolaus Schultz; Timothy A Chan; Simon N Powell; Jorge S Reis-Filho; Christine A Iacobuzio-Donahue
Journal:  Clin Cancer Res       Date:  2020-05-22       Impact factor: 12.531

Review 2.  Biomarker-Guided Development of DNA Repair Inhibitors.

Authors:  James M Cleary; Andrew J Aguirre; Geoffrey I Shapiro; Alan D D'Andrea
Journal:  Mol Cell       Date:  2020-05-26       Impact factor: 17.970

3.  Combination Therapy with Radiation and PARP Inhibition Enhances Responsiveness to Anti-PD-1 Therapy in Colorectal Tumor Models.

Authors:  Steven N Seyedin; M M Hasibuzzaman; Vivan Pham; Michael S Petronek; Cameron Callaghan; Amanda L Kalen; Kranti A Mapuskar; Sarah L Mott; Douglas R Spitz; Bryan G Allen; Joseph M Caster
Journal:  Int J Radiat Oncol Biol Phys       Date:  2020-02-06       Impact factor: 7.038

Review 4.  Targeting the DNA damage response in immuno-oncology: developments and opportunities.

Authors:  Roman M Chabanon; Mathieu Rouanne; Christopher J Lord; Jean-Charles Soria; Philippe Pasero; Sophie Postel-Vinay
Journal:  Nat Rev Cancer       Date:  2021-08-10       Impact factor: 60.716

5.  Targeting DNA Repair to Drive Immune Responses: It's Time to Reconsider the Strategy for Clinical Translation.

Authors:  Nobuyuki Takahashi; Ira Surolia; Anish Thomas
Journal:  Clin Cancer Res       Date:  2020-02-17       Impact factor: 12.531

Review 6.  Poly (ADP-ribose) Polymerase Inhibition in Patients with Breast Cancer and BRCA 1 and 2 Mutations.

Authors:  Yolanda Jerez; Ivan Márquez-Rodas; Inmaculada Aparicio; Manuel Alva; Miguel Martín; Sara López-Tarruella
Journal:  Drugs       Date:  2020-02       Impact factor: 9.546

Review 7.  Breast Cancer: A Molecularly Heterogenous Disease Needing Subtype-Specific Treatments.

Authors:  Ugo Testa; Germana Castelli; Elvira Pelosi
Journal:  Med Sci (Basel)       Date:  2020-03-23

Review 8.  Novel combinatorial strategies for boosting the efficacy of immune checkpoint inhibitors in advanced breast cancers.

Authors:  M F Tolba; H Elghazaly; E Bousoik; M M A Elmazar; S M Tolaney
Journal:  Clin Transl Oncol       Date:  2021-04-19       Impact factor: 3.405

Review 9.  DNA damage repair: historical perspectives, mechanistic pathways and clinical translation for targeted cancer therapy.

Authors:  Ruixue Huang; Ping-Kun Zhou
Journal:  Signal Transduct Target Ther       Date:  2021-07-09

10.  Niraparib for Advanced Breast Cancer with Germline BRCA1 and BRCA2 Mutations: the EORTC 1307-BCG/BIG5-13/TESARO PR-30-50-10-C BRAVO Study.

Authors:  Nicholas C Turner; Judith Balmaña; Coralie Poncet; Theodora Goulioti; Konstantinos Tryfonidis; Aafke H Honkoop; Gabriele Zoppoli; Evangelia Razis; Oskar T Johannsson; Marco Colleoni; Andrew N Tutt; William Audeh; Michail Ignatiadis; Audrey Mailliez; Olivier Trédan; Antonino Musolino; Peter Vuylsteke; Maria Jose Juan-Fita; Iain R J Macpherson; Bella Kaufman; Luis Manso; Lori J Goldstein; Susan L Ellard; István Láng; Kai Yu Jen; Virginie Adam; Saskia Litière; John Erban; David A Cameron
Journal:  Clin Cancer Res       Date:  2021-07-22       Impact factor: 12.531

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