Literature DB >> 31183658

Respiratory Complications in Patients with Hyper IgM Syndrome.

Bobak Moazzami1, Reza Yazdani2, Gholamreza Azizi3, Fatemeh Kiaei1, Mitra Tafakori1, Mohammadreza Modaresi4, Rohola Shirzadi4, Seyed Alireza Mahdaviani5, Mahsa Sohani6, Hassan Abolhassani1,7, Asghar Aghamohammadi8.   

Abstract

PURPOSE: Hyper Immunoglobulin M (HIgM) syndrome is a heterogeneous group of primary immunodeficiency disorders, characterized by recurrent infections and associated with decreased serum IgG and IgA, but normal or increased IgM. The aim of the present study was to evaluate respiratory manifestations in patients with HIgM syndrome.
METHODS: A total number of 62 patients, including 46 males and 16 females were included in the present study. To investigate the respiratory complications among HIgM patients, we evaluated the clinical hospital records, immunologic and molecular diagnostic assays, pulmonary function tests (PFT), and high-resolution computed tomography (HRCT) scans.
RESULTS: Pneumonia was the most common respiratory manifestation (n = 35, 56.4%), followed by otitis media (45.1%), sinusitis (33.8%), and bronchiectasis (14.5%). 52.1% of the patients had abnormal PFT results, with a predominant restrictive pattern of changes. HRCT scans demonstrated abnormal findings in 85.7% of patients with found mutations. Ten cases had hilar lymphadenopathy and para-hilar infiltrates in their HRCT findings. Genetic diagnosis was confirmed in 29 HIgM patients (72.4% CD40 ligand (CD40L) and 24.1% activation-induced cytidine deaminase (AICDA/AID) deficiencies). Majority of patients with CD40L (71.4%) and AID (57.1%) deficiencies had missense mutations. Pneumonia and abnormal high-resolution computed tomography (HRCT) findings were more frequent among patients with CD40L mutation. Respiratory failure constituted the major cause of mortality (37.5%) with majority of cases occurring in CD40L-deficient patients (50%).
CONCLUSIONS: Respiratory complications are common in patients with HIgM syndrome. A proper awareness of respiratory manifestations in patients with HIgM may result in improved management, reduced morbidity and mortality, and an improvement in the quality of life of the patients.

Entities:  

Keywords:  Hyper immunoglobulin M; bronchiectasis; otitis media; pneumonia; primary immunodeficiency; respiratory complications; sinusitis

Mesh:

Substances:

Year:  2019        PMID: 31183658     DOI: 10.1007/s10875-019-00650-3

Source DB:  PubMed          Journal:  J Clin Immunol        ISSN: 0271-9142            Impact factor:   8.317


  51 in total

1.  Early and prolonged intravenous immunoglobulin replacement therapy in childhood agammaglobulinemia: a retrospective survey of 31 patients.

Authors:  P Quartier; M Debré; J De Blic; R de Sauverzac; N Sayegh; N Jabado; E Haddad; S Blanche; J L Casanova; C I Smith; F Le Deist; G de Saint Basile; A Fischer
Journal:  J Pediatr       Date:  1999-05       Impact factor: 4.406

2.  The health status and quality of life of adults with X-linked agammaglobulinemia.

Authors:  Vanessa Howard; Jeffrey M Greene; Savita Pahwa; Jerry A Winkelstein; John M Boyle; Mehmet Kocak; Mary Ellen Conley
Journal:  Clin Immunol       Date:  2005-12-22       Impact factor: 3.969

3.  Pulmonary complications in primary hypogammaglobulinemia: a survey by high resolution CT scan.

Authors:  M Gharagozlou; F A Ebrahimi; A Farhoudi; A Aghamohammadi; M-H Bemanian; Z Chavoshzadeh; M Heidarzadeh; M Mehdizadeh; M Moin; M Movahedi; M Nabavi; Z Pourpak; N Rezaei
Journal:  Monaldi Arch Chest Dis       Date:  2006-06

Review 4.  Hyper immunoglobulin M syndrome due to CD40 deficiency: clinical, molecular, and immunological features.

Authors:  Vassilios Lougaris; Raffaele Badolato; Simona Ferrari; Alessandro Plebani
Journal:  Immunol Rev       Date:  2005-02       Impact factor: 12.988

Review 5.  Immunodeficiency with hyper-IgM (HIM).

Authors:  L D Notarangelo; M Duse; A G Ugazio
Journal:  Immunodefic Rev       Date:  1992

6.  Specific missense mutations in NEMO result in hyper-IgM syndrome with hypohydrotic ectodermal dysplasia.

Authors:  A Jain; C A Ma; S Liu; M Brown; J Cohen; W Strober
Journal:  Nat Immunol       Date:  2001-03       Impact factor: 25.606

7.  Mutations in activation-induced cytidine deaminase in patients with hyper IgM syndrome.

Authors:  Y Minegishi; A Lavoie; C Cunningham-Rundles; P M Bédard; J Hébert; L Côté; K Dan; D Sedlak; R H Buckley; A Fischer; A Durandy; M E Conley
Journal:  Clin Immunol       Date:  2000-12       Impact factor: 3.969

8.  The X-linked hyper-IgM syndrome: clinical and immunologic features of 79 patients.

Authors:  Jerry A Winkelstein; Mary C Marino; Hans Ochs; Ramsey Fuleihan; Paul R Scholl; Raif Geha; E Richard Stiehm; Mary Ellen Conley
Journal:  Medicine (Baltimore)       Date:  2003-11       Impact factor: 1.889

9.  Pulmonary and sinus diseases in primary humoral immunodeficiencies with chronic productive cough.

Authors:  F Rusconi; C Panisi; R M Dellepiane; F Cardinale; L Chini; B Martire; N Bonelli; G Felisati; M C Pietrogrande
Journal:  Arch Dis Child       Date:  2003-12       Impact factor: 3.791

10.  Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the Hyper-IgM syndrome (HIGM2).

Authors:  P Revy; T Muto; Y Levy; F Geissmann; A Plebani; O Sanal; N Catalan; M Forveille; R Dufourcq-Labelouse; A Gennery; I Tezcan; F Ersoy; H Kayserili; A G Ugazio; N Brousse; M Muramatsu; L D Notarangelo; K Kinoshita; T Honjo; A Fischer; A Durandy
Journal:  Cell       Date:  2000-09-01       Impact factor: 41.582

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  1 in total

Review 1.  Imaging Features of Primary Immunodeficiency Disorders.

Authors:  Jose A Rodriguez; Tami J Bang; Carlos S Restrepo; Daniel B Green; Lorna P Browne; Daniel Vargas
Journal:  Radiol Cardiothorac Imaging       Date:  2021-03-25
  1 in total

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