Literature DB >> 31144522

Loss of appetite is associated with a loss of weight and fat mass in patients with amyotrophic lateral sclerosis.

Shyuan T Ngo1,2,3,4,5, Ruben P A van Eijk6,7, V Chachay8, Leonard H van den Berg6, Pamela A McCombe3,4,5, Robert D Henderson2,3,4,5, Frederik J Steyn1,3,4,5.   

Abstract

Objective: Weight loss in amyotrophic lateral sclerosis (ALS) is associated with faster disease progression and shorter survival. It has different possible causes, including loss of appetite. Our objective is to determine the prevalence and impact of loss of appetite on change in body weight and composition in patients with ALS.
Methods: We conducted a prospective case-control study, comparing demographic, clinical, appetite and prognostic features between 62 patients with ALS and 45 healthy non-neurodegenerative disease (NND) controls. To determine the impact of loss of appetite on weight throughout disease course, we conducted serial assessments at ∼three to four-month intervals.
Results: Loss of appetite is more prevalent in patients with ALS than NND controls (29 vs. 11.1%, odds ratio = 3.27 (1.1-9.6); p < 0.01). In patients with ALS, loss of appetite is associated with greater weight loss and greater loss of fat mass. Appetite scores in patients with ALS worsens as disease progresses and are correlated with worsening ALS Functional Rating Scale-Revised scores.
Conclusion: We confirm that loss of appetite is prevalent in patients with ALS and is significantly associated with weight loss and loss of fat mass. Appetite worsens with disease progression. Identification and early interventions to address loss of appetite in patients with ALS may prevent or slow weight loss; this could improve disease outcome.

Entities:  

Year:  2019        PMID: 31144522     DOI: 10.1080/21678421.2019.1621346

Source DB:  PubMed          Journal:  Amyotroph Lateral Scler Frontotemporal Degener        ISSN: 2167-8421            Impact factor:   4.092


  9 in total

Review 1.  Altered Bioenergetics and Metabolic Homeostasis in Amyotrophic Lateral Sclerosis.

Authors:  Andrew T Nelson; Davide Trotti
Journal:  Neurotherapeutics       Date:  2022-06-30       Impact factor: 6.088

2.  The feeding behaviour of Amyotrophic Lateral Sclerosis mouse models is modulated by the Ca2+ -activated KCa 3.1 channels.

Authors:  Germana Cocozza; Stefano Garofalo; Marta Morotti; Giuseppina Chece; Alfonso Grimaldi; Mario Lecce; Ferdinando Scavizzi; Rossella Menghini; Viviana Casagrande; Massimo Federici; Marcello Raspa; Heike Wulff; Cristina Limatola
Journal:  Br J Pharmacol       Date:  2021-10-05       Impact factor: 9.473

3.  Fat mass loss correlates with faster disease progression in amyotrophic lateral sclerosis patients: Exploring the utility of dual-energy x-ray absorptiometry in a prospective study.

Authors:  Ikjae Lee; Mohamed Kazamel; Tarrant McPherson; Jeremy McAdam; Marcas Bamman; Amy Amara; Daniel L Smith; Peter H King
Journal:  PLoS One       Date:  2021-05-06       Impact factor: 3.240

Review 4.  Potential Preventive Strategies for Amyotrophic Lateral Sclerosis.

Authors:  B Kuraszkiewicz; H Goszczyńska; T Podsiadły-Marczykowska; M Piotrkiewicz; P Andersen; M Gromicho; J Grosskreutz; M Kuźma-Kozakiewicz; S Petri; B Stubbendorf; K Szacka; H Uysal; M de Carvalho
Journal:  Front Neurosci       Date:  2020-05-26       Impact factor: 4.677

Review 5.  Existing and Emerging Metabolomic Tools for ALS Research.

Authors:  Christine Germeys; Tijs Vandoorne; Valérie Bercier; Ludo Van Den Bosch
Journal:  Genes (Basel)       Date:  2019-12-05       Impact factor: 4.096

Review 6.  CNS glucose metabolism in Amyotrophic Lateral Sclerosis: a therapeutic target?

Authors:  Tesfaye Wolde Tefera; Frederik J Steyn; Shyuan T Ngo; Karin Borges
Journal:  Cell Biosci       Date:  2021-01-11       Impact factor: 7.133

7.  Loss of appetite in patients with amyotrophic lateral sclerosis is associated with weight loss and anxiety/depression.

Authors:  Yajun Wang; Shan Ye; Lu Chen; Lu Tang; Dongsheng Fan
Journal:  Sci Rep       Date:  2021-04-27       Impact factor: 4.379

8.  The Small Molecule PPARγ Agonist GL516 Induces Feeding-Stimulatory Effects in Hypothalamus Cells Hypo-E22 and Isolated Hypothalami.

Authors:  Annalisa Chiavaroli; Viviana di Giacomo; Barbara De Filippis; Amelia Cataldi; Claudio Ferrante; Letizia Giampietro
Journal:  Molecules       Date:  2022-07-30       Impact factor: 4.927

9.  Correlation of weight and body composition with disease progression rate in patients with amyotrophic lateral sclerosis.

Authors:  Jin-Yue Li; Xiao-Han Sun; Zheng-Yi Cai; Dong-Chao Shen; Xun-Zhe Yang; Ming-Sheng Liu; Li-Ying Cui
Journal:  Sci Rep       Date:  2022-08-02       Impact factor: 4.996

  9 in total

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