Literature DB >> 31125419

Predicting Hemolytic Uremic Syndrome and Renal Replacement Therapy in Shiga Toxin-producing Escherichia coli-infected Children.

Ryan S McKee1, David Schnadower2, Phillip I Tarr3, Jianling Xie4, Yaron Finkelstein5, Neil Desai6, Roni D Lane7, Kelly R Bergmann8, Ron L Kaplan9, Selena Hariharan3, Andrea T Cruz10, Daniel M Cohen11, Andrew Dixon12, Sriram Ramgopal13, Annie Rominger14, Elizabeth C Powell15, Jennifer Kilgar16, Kenneth A Michelson17, Darcy Beer6, Martin Bitzan18, Christopher M Pruitt19, Kenneth Yen20, Garth D Meckler21, Amy C Plint22, Stuart Bradin23, Thomas J Abramo24, Serge Gouin25, April J Kam26, Abigail Schuh27, Fran Balamuth28, Tracy E Hunley29, John T Kanegaye30,31, Nicholas E Jones32, Usha Avva33, Robert Porter34, Daniel M Fein35, Jeffrey P Louie36, Stephen B Freedman37.   

Abstract

BACKGROUND: Shiga toxin-producing Escherichia coli (STEC) infections are leading causes of pediatric acute renal failure. Identifying hemolytic uremic syndrome (HUS) risk factors is needed to guide care.
METHODS: We conducted a multicenter, historical cohort study to identify features associated with development of HUS (primary outcome) and need for renal replacement therapy (RRT) (secondary outcome) in STEC-infected children without HUS at initial presentation. Children aged <18 years who submitted STEC-positive specimens between January 2011 and December 2015 at a participating study institution were eligible.
RESULTS: Of 927 STEC-infected children, 41 (4.4%) had HUS at presentation; of the remaining 886, 126 (14.2%) developed HUS. Predictors (all shown as odds ratio [OR] with 95% confidence interval [CI]) of HUS included younger age (0.77 [.69-.85] per year), leukocyte count ≥13.0 × 103/μL (2.54 [1.42-4.54]), higher hematocrit (1.83 [1.21-2.77] per 5% increase) and serum creatinine (10.82 [1.49-78.69] per 1 mg/dL increase), platelet count <250 × 103/μL (1.92 [1.02-3.60]), lower serum sodium (1.12 [1.02-1.23 per 1 mmol/L decrease), and intravenous fluid administration initiated ≥4 days following diarrhea onset (2.50 [1.14-5.46]). A longer interval from diarrhea onset to index visit was associated with reduced HUS risk (OR, 0.70 [95% CI, .54-.90]). RRT predictors (all shown as OR [95% CI]) included female sex (2.27 [1.14-4.50]), younger age (0.83 [.74-.92] per year), lower serum sodium (1.15 [1.04-1.27] per mmol/L decrease), higher leukocyte count ≥13.0 × 103/μL (2.35 [1.17-4.72]) and creatinine (7.75 [1.20-50.16] per 1 mg/dL increase) concentrations, and initial intravenous fluid administration ≥4 days following diarrhea onset (2.71 [1.18-6.21]).
CONCLUSIONS: The complex nature of STEC infection renders predicting its course a challenge. Risk factors we identified highlight the importance of avoiding dehydration and performing close clinical and laboratory monitoring.
© The Author(s) 2019. Published by Oxford University Press for the Infectious Diseases Society of America. All rights reserved. For permissions, e-mail: journals.permissions@oup.com.

Entities:  

Keywords:  Shiga-toxigenic Escherichia coli; child; emergency service; hemolytic uremic syndrome; renal replacement therapy

Mesh:

Year:  2020        PMID: 31125419      PMCID: PMC7931832          DOI: 10.1093/cid/ciz432

Source DB:  PubMed          Journal:  Clin Infect Dis        ISSN: 1058-4838            Impact factor:   9.079


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