Literature DB >> 31092713

Neutrophil Extracellular Traps Profiles in Patients with Incident Systemic Lupus Erythematosus and Lupus Nephritis.

Maurizio Bruschi1,2, Alice Bonanni1,2, Andrea Petretto1,2, Augusto Vaglio1,2, Federico Pratesi1,2, Laura Santucci1,2, Paola Migliorini1,2, Roberta Bertelli1,2, Maricla Galetti1,2, Silvana Belletti1,2, Lorenzo Cavagna1,2, Gabriella Moroni1,2, Franco Franceschini1,2, Micaela Fredi1,2, Giulia Pazzola1,2, Landino Allegri1,2, Renato Alberto Sinico1,2, Giampaola Pesce1,2, Marcello Bagnasco1,2, Angelo Manfredi1,2, Giuseppe A Ramirez1,2, Paola Ramoino1,2, Paolo Bianchini1,2, Francesco Puppo1,2, Francesca Pupo1,2, Simone Negrini1,2, Federico Mattana1,2, Giacomo Emmi1,2, Giacomo Garibotto1,2, Domenico Santoro1,2, Francesco Scolari1,2, Angelo Ravelli1,2, Angela Tincani1,2, Paolo Cravedi1,2, Stefano Volpi1,2, Giovanni Candiano1,2, Gian Marco Ghiggeri3,4.   

Abstract

OBJECTIVE: Neutrophil extracellular traps (NET) expose modified antigens for autoantibodies in vasculitis. Little is known about levels and removal pathways of NET in systemic lupus erythematosus (SLE), especially in lupus nephritis (LN). We determined circulating levels and defined NET removal in large subsets of patients with incident SLE (iSLE), some of whom had new-onset nephritis.
METHODS: Serum levels of NET (ELISA), DNase1/DNase1L3 (ELISA), and DNase activity (functional assay) were determined in 216 patients with iSLE [103 had incident LN (iLN)], in 50 patients with other primary glomerulonephritis, and in healthy controls. Ex vivo NET production by neutrophils purified from a random selection of patients was quantified as elastase/DNA release and by immunofluorescence techniques.
RESULTS: Serum NET levels were very high in iSLE/iLN compared to all groups of controls and correlated with anti-dsDNA, C3-C4, and proteinuria; iLN had the highest levels. DNase activity was decreased in iLN compared to SLE (20% had one-half DNase activity) despite similar serum levels of DNase1/DNase1L3. In these cases, pretreatment of serum with protein A restored DNase efficiency; 1 patient was homozygous for a c.289_290delAC variant of DNASE1L3. Ex vivo NET production by neutrophils purified from LN, SLE, and normal controls was similar in all cases.
CONCLUSION: Patients with iLN have increased circulating NET and reduced DNase activity, the latter being explained by the presence of inhibitory substances in circulation and/or by rare DNase1L3 mutations. Accumulation of NET derives from a multifactorial mechanism, and is associated and may contribute to disease severity in SLE, in particular to renal lesions. (Clinical trial registration: The Zeus study was registered at ClinicalTrials.gov, study number NCT02403115).

Entities:  

Keywords:  DNASE ACTIVITY; DNASE LEVEL; DNASE MUTATIONS; NEUTROPHIL EXTRACELLULAR TRAPS

Mesh:

Substances:

Year:  2019        PMID: 31092713      PMCID: PMC6917988          DOI: 10.3899/jrheum.181232

Source DB:  PubMed          Journal:  J Rheumatol        ISSN: 0315-162X            Impact factor:   4.666


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