Literature DB >> 31011798

Olfactory anosognosia is a predictor of cognitive decline and dementia conversion in Parkinson's disease.

Han Soo Yoo1, Seok Jong Chung1, Yang Hyun Lee1, Byoung Seok Ye1, Young H Sohn1, Phil Hyu Lee2,3.   

Abstract

OBJECTIVE: Parkinson's disease (PD) patients are often unaware of olfactory deficits despite having hyposmia from the early stages. We aimed to evaluate whether olfactory anosognosia is a predictor of cognitive decline in PD.
METHODS: In this retrospective cohort study, we recruited 77 PD patients who underwent both olfactory and neuropsychological tests and were followed-up for over 5 years. Based on the degree of olfactory dysfunction and awareness of its presence, patients were classified as normosmic patients (Normosmia group, n = 15), hyposmic patients without olfactory anosognosia (Hyposmia-OA-, n = 40), or hyposmic patients with olfactory anosognosia (Hyposmia-OA+, n = 22). We compared the rates of cognitive decline using linear mixed model and dementia conversion using a survival analysis among the groups.
RESULTS: A higher proportion of patients in the Hyposmia-OA+ group had mild cognitive impairment at baseline (77.3%) and dementia converter at follow-up (50.0%). The Hyposmia-OA+ group exhibited a faster decline in frontal executive and global cognitive function than did the Normosmia and Hyposmia-OA- groups. A Kaplan-Meier analysis demonstrated that the conversion rate to dementia was significantly higher in the Hyposmia-OA+ group than in the Normosmia (P = 0.007) and Hyposmia-OA- (P = 0.038) groups. A Cox regression analysis showed that olfactory anosognosia remained a significant predictor of time to develop dementia in the Hyposmia-OA+ group compared to the Normosmia group (adjusted hazard ratio 3.30; 95% confidence interval 1.10-8.21).
CONCLUSION: This study suggests that olfactory anosognosia is a predictor of cognitive decline and dementia conversion in PD.

Entities:  

Keywords:  Anosognosia; Cognitive decline; Dementia; Olfactory dysfunction; Parkinson’s disease

Year:  2019        PMID: 31011798     DOI: 10.1007/s00415-019-09297-x

Source DB:  PubMed          Journal:  J Neurol        ISSN: 0340-5354            Impact factor:   4.849


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