Literature DB >> 31008320

Arterial branching and basal ganglia lacunes: A study in pure small vessel disease.

Fiona C Moreton1, Marco Düring2, Thanh Phan3, Velandai Srikanth3, Richard Beare3, Xuya Huang1, Eric Jouvent4, Hugues Chabriat4, Martin Dichgans2, Keith W Muir1.   

Abstract

INTRODUCTION: Lacunes are defined morphologically by size and location, but radiological characteristics alone may be unable to distinguish small vessel disease aetiology from alternative mechanisms. We investigated the branching order of arterial vessels associated with basal ganglia lacunes in cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL), in order to improve the understanding of their pathogenesis in pure cerebral small vessel disease. PATIENTS AND METHODS: Adults with a confirmed diagnosis of CADASIL were included. A pilot study was conducted in a Scottish CADASIL cohort. The Paris-Munich CADASIL cohort was used for independent validation. Lacunes identified on T1-weighted magnetic resonance imaging scans were registered to a standard brain template. A microangiographic template of the basal ganglia vasculature was automatically overlaid onto coronal slices, and raters estimated the vessel branching order related to each lacune.
RESULTS: Of 179 lacunes, 150 (84%) were associated with third-order vessels. In 14 incident lacunes, 11 (79%) were associated with third-order vessels. In the pilot study, lacune volume was significantly lower in lacunes associated with third-order vessels (0.04 ml ± 0.04 ml) compared to second-order vessels (0.48 ± 0.16 ml; p < 0.001). DISCUSSION: In this study of CADASIL patients, most lacunes were small and associated with third-order vessel disease. This suggests that these are the vessels primarily affected in cerebral small vessel disease. Microangiographic template techniques could be used to further investigate in a general stroke population whether finding large lacunes originating from higher order vessels indicates an alternative cause of stroke.
CONCLUSION: Lacunes in pure small vessel disease are associated with the smallest vessels in the basal ganglia.

Entities:  

Keywords:  Infarction; cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy; lacunes; magnetic resonance imaging

Year:  2017        PMID: 31008320      PMCID: PMC6454827          DOI: 10.1177/2396987317718450

Source DB:  PubMed          Journal:  Eur Stroke J        ISSN: 2396-9873


  23 in total

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2.  A global optimisation method for robust affine registration of brain images.

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6.  Tertiary microvascular territories define lacunar infarcts in the basal ganglia.

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Journal:  Neuroimage       Date:  2004       Impact factor: 6.556

8.  Diffusion-weighted imaging identifies a subset of lacunar infarction associated with embolic source.

Authors:  H Ay; J Oliveira-Filho; F S Buonanno; M Ezzeddine; P W Schaefer; G Rordorf; L H Schwamm; R G Gonzalez; W J Koroshetz
Journal:  Stroke       Date:  1999-12       Impact factor: 7.914

9.  Classification and natural history of clinically identifiable subtypes of cerebral infarction.

Authors:  J Bamford; P Sandercock; M Dennis; J Burn; C Warlow
Journal:  Lancet       Date:  1991-06-22       Impact factor: 79.321

10.  Shape and volume of lacunar infarcts: a 3D MRI study in cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy.

Authors:  Dominique Hervé; Jean-François Mangin; Nicolas Molko; Marie-Germaine Bousser; Hugues Chabriat
Journal:  Stroke       Date:  2005-10-13       Impact factor: 7.914

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