| Literature DB >> 30955816 |
Tianqi Yu1, Yingnan Ding1, Yina Zhang2, Yulan Liu1, Yahui Li1, Jing Lei1, Jiyong Zhou2, Suquan Song1, Boli Hu3.
1. MOE International Joint Collaborative Research Laboratory for Animal Health and Food Safety, Institute of Immunology and College of Veterianry Medicine, Nanjing Agricultural University, Nanjing, China.
2. MOA Key Laboratory of Animal Virology, Department of Veterinary Medicine, Zhejiang University, Hangzhou, China.
3. MOE International Joint Collaborative Research Laboratory for Animal Health and Food Safety, Institute of Immunology and College of Veterianry Medicine, Nanjing Agricultural University, Nanjing, China. Electronic address: bolihu@zju.edu.cn.
Abstract
Entities:
Keywords: Autophagy; H1N1; Replication; circ-GATAD2A
Mesh:
A549 Cells
Autophagy/genetics
Class III Phosphatidylinositol 3-Kinases/genetics
GATA Transcription Factors/genetics
Gene Knockdown Techniques
Host Microbial Interactions/genetics
Humans
Influenza A Virus, H1N1 Subtype/genetics
Influenza A Virus, H1N1 Subtype/physiology
Influenza, Human/virology
RNA/genetics
RNA, Circular
Up-Regulation
Virus Replication
Substances:
GATA Transcription Factors
GATAD2A protein, human
RNA, Circular
RNA
Class III Phosphatidylinositol 3-Kinases
Year: 2019 PMID: 30955816 DOI: 10.1016/j.vetmic.2019.03.012
Source DB: PubMed Journal: Vet Microbiol ISSN: 0378-1135 Impact factor: 3.293