Literature DB >> 30944910

Adenosine Kinase Deficiency Increases Susceptibility to a Carcinogen.

Rkia El-Kharrag1, Randy Owen1, Detlev Boison1.   

Abstract

Background: Adenosine kinase (ADK) is a key regulator of hepatic metabolism. Its deficiency in the liver causes hepatic steatosis and methylation defects. In this study, we investigated whether reduced ADK expression affects the susceptibility of the liver to a carcinogen.
Methods: We investigated ADK expression in samples from 11 liver cancer patients. We used transgenic Adk-tg mice with reduced hepatic ADK to study their susceptibility to a carcinogen. We exposed 45 Adk-tg and 21 wild-type (WT) mice to the carcinogen diethylnitrosamine (DEN) and the tumor promoter phenobarbital (PB) and examined the survival and body weight.
Results: Seven of 11 patients with liver cancer had reduced ADK expression. A Kaplan-Meier survival curve showed a significantly increased mortality rate of DEN/PB-exposed Adk-tg mice compared with WT mice. Conclusions: Reduced hepatic ADK increases the susceptibility to the acute toxic effects of a carcinogen. Low hepatic ADK might be a risk factor and biomarker for cancer development.

Entities:  

Keywords:  adenosine kinase; carcinogen; hepatic toxicity; liver; metabolism; methylation

Year:  2019        PMID: 30944910      PMCID: PMC6444920          DOI: 10.1089/caff.2018.0019

Source DB:  PubMed          Journal:  J Caffeine Adenosine Res        ISSN: 2573-3397


  38 in total

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