Literature DB >> 30905651

Effects of 2-Aminoethyl Diphenylborinate, a Modulator of Transient Receptor Potential and Orai Channels in Subarachnoid Hemorrhage: An Experimental Study.

Mehmet Gazi Boyacı1, Usame Rakip2, Adem Aslan3, Halit Buğra Koca4, Esra Aslan5, Serhat Korkmaz3, Serhat Yıldızhan3.   

Abstract

BACKGROUND: Cerebral vasospasm remains a serious problem affecting morbidity and mortality in patients with subarachnoid hemorrhage (SAH) during neurosurgery. We aimed to demonstrate the role of the transient receptor potential channel and other channels for Ca2+ in the etiology of cerebral vasospasm using 2-aminoethyl diphenylborinate (2-APB) and the effective dose range of an unstudied pharmacological agent, which can limit vasospasm.
METHODS: We performed an experimental study using 32 Sprague-Dawley rats divided into 4 groups: sham group (n = 8), SAH group (n = 8), 2-APB group (SAH rats intraperitoneally administered with 0.5 mg/kg 2-APB; n = 8), and 2-APB-2 group (SAH rats intraperitoneally administered with 2 mg/kg 2-APB; n = 8). The rats were sacrificed after 24 hours, and superoxide dismutase, glutathione peroxidase, malondialdehyde, tumor necrosis factor-α, and interleukin-1β in the brain tissue and serum were measured. The histopathological investigation of brain tissue included measurement of the luminal diameter and wall thickness of the basilar artery (BA), and apoptotic cells in the hippocampus were counted after caspase staining.
RESULTS: Autologous arterial blood injection into the cisterna magna caused vasospasm in rats. 2-APB treatment increased the BA wall thickness and reduced the BA lumen diameter, inducing significant vascular changes. 2-APB also alleviated cell apoptosis at 24 hours after SAH.
CONCLUSION: In experimental SAH in rats, 2-APB treatment increased the BA wall thickness and reduced the BA lumen diameter, inducing significant vascular changes. 2-APB also alleviated cell apoptosis at 24 hours after SAH.
Copyright © 2019 Elsevier Inc. All rights reserved.

Entities:  

Keywords:  2-Aminoethyl diphenylborate; Ca(2+) channels; Cerebral vasospasm; Orai channels; Subarachnoid hemorrhage; TRP channels

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Year:  2019        PMID: 30905651     DOI: 10.1016/j.wneu.2019.03.129

Source DB:  PubMed          Journal:  World Neurosurg        ISSN: 1878-8750            Impact factor:   2.104


  2 in total

Review 1.  The mechanism and relevant mediators associated with neuronal apoptosis and potential therapeutic targets in subarachnoid hemorrhage.

Authors:  Qi Tian; Sheng Liu; Shou-Meng Han; Wei Zhang; Xian-Yao Qin; Jun-Hui Chen; Cheng-Li Liu; Yu-Jia Guo; Ming-Chang Li
Journal:  Neural Regen Res       Date:  2023-02       Impact factor: 6.058

2.  The Transient Receptor Potential Melastatin 7 (TRPM7) Inhibitors Suppress Seizure-Induced Neuron Death by Inhibiting Zinc Neurotoxicity.

Authors:  Jeong Hyun Jeong; Song Hee Lee; A Ra Kho; Dae Ki Hong; Dong Hyeon Kang; Beom Seok Kang; Min Kyu Park; Bo Young Choi; Hui Chul Choi; Man-Sup Lim; Sang Won Suh
Journal:  Int J Mol Sci       Date:  2020-10-24       Impact factor: 5.923

  2 in total

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