Literature DB >> 30877299

Hypertonic Saline is Superior to Mannitol for the Combined Effect on Intracranial Pressure and Cerebral Perfusion Pressure Burdens in Patients With Severe Traumatic Brain Injury.

Halinder S Mangat1,2, Xian Wu3, Linda M Gerber3, Justin T Schwarz2,4, Malik Fakhar1, Santosh B Murthy1, Philip E Stieg2, Jamshid Ghajar5,6, Roger Härtl2.   

Abstract

BACKGROUND: Hypertonic saline (HTS) and mannitol are effective in reducing intracranial pressure (ICP) after severe traumatic brain injury (TBI). However, their simultaneous effect on the cerebral perfusion pressure (CPP) and ICP has not been studied rigorously.
OBJECTIVE: To determine the difference in effects of HTS and mannitol on the combined burden of high ICP and low CPP in patients with severe TBI.
METHODS: We performed a case-control study using prospectively collected data from the New York State TBI-trac® database (Brain Trauma Foundation, New York, New York). Patients who received only 1 hyperosmotic agent, either mannitol or HTS for raised ICP, were included. Patients in the 2 groups were matched (1:1 and 1:2) for factors associated with 2-wk mortality: age, Glasgow Coma Scale score, pupillary reactivity, hypotension, abnormal computed tomography scans, and craniotomy. Primary endpoint was the combined burden of ICPhigh (> 25 mm Hg) and CPPlow (< 60 mm Hg).
RESULTS: There were 25 matched pairs for 1:1 comparison and 24 HTS patients matched to 48 mannitol patients in 1:2 comparisons. Cumulative median osmolar doses in the 2 groups were similar. In patients treated with HTS compared to mannitol, total number of days (0.6 ± 0.8 vs 2.4 ± 2.3 d, P < .01), percentage of days with (8.8 ± 10.6 vs 28.1 ± 26.9%, P < .01), and the total duration of ICPhigh + CPPlow (11.12 ± 14.11 vs 30.56 ± 31.89 h, P = .01) were significantly lower. These results were replicated in the 1:2 match comparisons.
CONCLUSION: HTS bolus therapy appears to be superior to mannitol in reduction of the combined burden of intracranial hypertension and associated hypoperfusion in severe TBI patients.
Copyright © 2019 by the Congress of Neurological Surgeons.

Entities:  

Keywords:  Cerebral perfusion pressure; Hypertonic saline; Intracranial pressure; Mannitol; Traumatic brain injury

Mesh:

Substances:

Year:  2020        PMID: 30877299      PMCID: PMC8253301          DOI: 10.1093/neuros/nyz046

Source DB:  PubMed          Journal:  Neurosurgery        ISSN: 0148-396X            Impact factor:   4.654


  43 in total

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Authors:  Susan L Bratton; Randall M Chestnut; Jamshid Ghajar; Flora F McConnell Hammond; Odette A Harris; Roger Hartl; Geoffrey T Manley; Andrew Nemecek; David W Newell; Guy Rosenthal; Joost Schouten; Lori Shutter; Shelly D Timmons; Jamie S Ullman; Walter Videtta; Jack E Wilberger; David W Wright
Journal:  J Neurotrauma       Date:  2007       Impact factor: 5.269

2.  Intracranial pressure dose and outcome in traumatic brain injury.

Authors:  Kevin N Sheth; Deborah M Stein; Bizhan Aarabi; Peter Hu; Joseph A Kufera; Thomas M Scalea; Daniel F Hanley
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3.  Prevention of secondary ischemic insults after severe head injury.

Authors:  C S Robertson; A B Valadka; H J Hannay; C F Contant; S P Gopinath; M Cormio; M Uzura; R G Grossman
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Authors:  F Nath; S Galbraith
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5.  A trial of intracranial-pressure monitoring in traumatic brain injury.

Authors:  Randall M Chesnut; Nancy Temkin; Nancy Carney; Sureyya Dikmen; Carlos Rondina; Walter Videtta; Gustavo Petroni; Silvia Lujan; Jim Pridgeon; Jason Barber; Joan Machamer; Kelley Chaddock; Juanita M Celix; Marianna Cherner; Terence Hendrix
Journal:  N Engl J Med       Date:  2012-12-12       Impact factor: 91.245

6.  Mannitol decreases ICP but does not improve brain-tissue pO2 in severely head-injured patients with intracranial hypertension.

Authors:  R Härtl; T F Bardt; K L Kiening; A S Sarrafzadeh; G H Schneider; A W Unterberg
Journal:  Acta Neurochir Suppl       Date:  1997

7.  Effect of mannitol and hypertonic saline on cerebral oxygenation in patients with severe traumatic brain injury and refractory intracranial hypertension.

Authors:  M Oddo; J M Levine; S Frangos; E Carrera; E Maloney-Wilensky; J L Pascual; W A Kofke; S A Mayer; P D LeRoux
Journal:  J Neurol Neurosurg Psychiatry       Date:  2009-03-16       Impact factor: 10.154

8.  Methodology for the control of intracranial pressure with hypertonic mannitol.

Authors:  H E James
Journal:  Acta Neurochir (Wien)       Date:  1980       Impact factor: 2.216

9.  Isovolume hypertonic solutes (sodium chloride or mannitol) in the treatment of refractory posttraumatic intracranial hypertension: 2 mL/kg 7.5% saline is more effective than 2 mL/kg 20% mannitol.

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10.  COBI (COntinuous hyperosmolar therapy for traumatic Brain-Injured patients) trial protocol: a multicentre randomised open-label trial with blinded adjudication of primary outcome.

Authors:  Antoine Roquilly; Sigismond Lasocki; Jean Denis Moyer; Olivier Huet; Pierre François Perrigault; Claire Dahyot-Fizelier; Philippe Seguin; Tarek Sharshar; Thomas Geeraerts; Francis Remerand; Fanny Feuillet; Karim Asehnoune
Journal:  BMJ Open       Date:  2017-09-24       Impact factor: 2.692

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2.  Construction and Evaluation of Prognosis Prediction Model for Patients with Brain Contusion and Laceration Based on Machine Learning.

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4.  Effects of two different doses of 3% hypertonic saline with mannitol during decompressive craniectomy following traumatic brain injury: A prospective, controlled study.

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5.  Hypertonic lactate for the treatment of intracranial hypertension in patients with acute brain injury.

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6.  Hypertonic Saline Compared to Mannitol for the Management of Elevated Intracranial Pressure in Traumatic Brain Injury: A Meta-Analysis.

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7.  Cerebral Edema in Traumatic Brain Injury: a Historical Framework for Current Therapy.

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8.  Guidelines for the Acute Treatment of Cerebral Edema in Neurocritical Care Patients.

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Journal:  Neurocrit Care       Date:  2020-06       Impact factor: 3.210

9.  Formulating a Stable Mannitol Infusion while Maintaining Hyperosmolarity.

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10.  Equimolar doses of hypertonic agents (saline or mannitol) in the treatment of intracranial hypertension after severe traumatic brain injury.

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