Literature DB >> 30810512

Myocardial dysfunction as a predictor of the severity and mortality of hypoxic ischaemic encephalopathy in severe perinatal asphyxia: a case-control study.

Himani Bhasin1, Charu Kohli2.   

Abstract

Background: In perinatal asphyxia, hypoxia often leads to myocardial ischaemia. Few studies have assessed the degree of myocardial dysfunction in severely asphyxiated term neonates. Aim: To assess the extent of myocardial damage in newborns with severe perinatal asphyxia.
Methods: A case-control study was conducted in asphyxiated newborns with hypoxic ischaemic encephalopathy (HIE) and in controls who were term non-asphyxiated newborns. Total (T) creatinine kinase (CK), CK-MB, troponin-T and 12-lead electrocardiography (ECG) and echocardiography were performed in both groups within 24-48 h after birth. The proportions of asphyxiated neonates with myocardial dysfunction and its relationship between severity of HIE and immediate outcome was compared.
Results: Five of 23 asphyxiated neonates developed stage I, 10 stage II and eight stage III HIE. Serum levels of CK-T and CK-MB were raised in all 23 cases and troponin-T was raised in 13 (56.5%) HIE cases. ECG was abnormal in all cases and echocardiography in three (13%). Left ventricular ejection fraction (LVEF) and right ventricular ejection fraction (RVEF) were significantly decreased in all asphyxiated neonates. Eight (35%) patients died. Enzyme levels were higher and ECG and echocardiography abnormalities were common in infants with more severe HIE (p <0.05). Mean serum levels of CK-T and MB (p <0.001) and troponin-T (p =0.002) were higher in non-survivors. Mean LVEF and RVEF values were higher in survivors (p <0.001). All the controls had normal enzyme levels and echocardiography. ECG was abnormal in one control.
Conclusion: Cardiac enzymes, ECG and echocardiography changes were associated with increasing severity of HIE and mortality.

Entities:  

Keywords:  ECG; Perinatal asphyxia; creatinine kinase total and MB; echocardiography; hypoxic ischaemic encephalopathy; troponin-T

Mesh:

Substances:

Year:  2019        PMID: 30810512     DOI: 10.1080/20469047.2019.1581462

Source DB:  PubMed          Journal:  Paediatr Int Child Health        ISSN: 2046-9047            Impact factor:   1.990


  3 in total

1.  Which factors predict outcomes of neonates with hypoxic-ischemic encephalopathy following therapeutic hypothermia?

Authors:  Gyu Hong Shim
Journal:  Clin Exp Pediatr       Date:  2020-12-11

2.  Correlation of cardiac troponin T levels with inotrope requirement, hypoxic-ischemic encephalopathy, and survival in asphyxiated neonates.

Authors:  Ramesh Bhat Yellanthoor; Dineshkumar Rajamanickam
Journal:  World J Clin Pediatr       Date:  2022-01-09

3.  Effect of a Larger Flush Volume on Bioavailability and Efficacy of Umbilical Venous Epinephrine during Neonatal Resuscitation in Ovine Asphyxial Arrest.

Authors:  Deepika Sankaran; Payam Vali; Praveen Chandrasekharan; Peggy Chen; Sylvia F Gugino; Carmon Koenigsknecht; Justin Helman; Jayasree Nair; Bobby Mathew; Munmun Rawat; Lori Nielsen; Amy L Lesneski; Morgan E Hardie; Ziad Alhassen; Houssam M Joudi; Evan M Giusto; Lida Zeinali; Heather K Knych; Gary M Weiner; Satyan Lakshminrusimha
Journal:  Children (Basel)       Date:  2021-06-01
  3 in total

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