Literature DB >> 30789418

Protease-Sensitive Pancreatic Lipase Variants Are Associated With Early Onset Chronic Pancreatitis.

Denise Lasher1, András Szabó2,3, Atsushi Masamune4, Jian-Min Chen5, Xunjun Xiao6, David C Whitcomb7, M Michael Barmada8,9, Maren Ewers1, Claudia Ruffert10, Sumit Paliwal11,12, Prachand Issarapu11, Seema Bhaskar11, K Radha Mani11, Giriraj R Chandak11, Helmut Laumen1, Emmanuelle Masson5, Kiyoshi Kume4, Shin Hamada4, Eriko Nakano4, Katharina Seltsam10, Peter Bugert13, Thomas Müller14, David A Groneberg15, Tooru Shimosegawa4, Jonas Rosendahl10,16, Claude Férec5, Mark E Lowe6, Heiko Witt1, Miklós Sahin-Tóth2.   

Abstract

OBJECTIVES: Premature activation of the digestive protease trypsin within the pancreatic parenchyma is a critical factor in the pathogenesis of pancreatitis. Alterations in genes that affect intrapancreatic trypsin activity are associated with chronic pancreatitis (CP). Recently, carboxyl ester lipase emerged as a trypsin-independent risk gene. Here, we evaluated pancreatic lipase (PNLIP) as a potential novel susceptibility gene for CP.
METHODS: We analyzed all 13 PNLIP exons in 429 nonalcoholic patients with CP and 600 control subjects from Germany, in 632 patients and 957 controls from France, and in 223 patients and 1,070 controls from Japan by DNA sequencing. Additionally, we analyzed selected exons in further 545 patients with CP and 1,849 controls originating from Germany, United States, and India. We assessed the cellular secretion, lipase activity, and proteolytic stability of recombinant PNLIP variants.
RESULTS: In the German discovery cohort, 8/429 (1.9%) patients and 2/600 (0.3%) controls carried a PNLIP missense variant (P = 0.02, odds ratio [OR] = 5.7, 95% confidence interval [CI] = 1.1-38.9). Variants detected in patients were prone to proteolytic degradation by trypsin and chymotrypsin. In the French replication cohort, protease-sensitive variants were also enriched in patients with early-onset CP (5/632 [0.8%]) vs controls (1/957 [0.1%]) (P = 0.04, OR = 7.6, 95% CI = 0.9-172.9). In contrast, we detected no protease-sensitive variants in the non-European populations. In the combined European data, protease-sensitive variants were found in 13/1,163 cases (1.1%) and in 3/3,000 controls (0.1%) (OR = 11.3, 95% CI = 3.0-49.9, P < 0.0001).
CONCLUSIONS: Our data indicate that protease-sensitive PNLIP variants are novel genetic risk factors for the development of CP.

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Year:  2019        PMID: 30789418      PMCID: PMC6624845          DOI: 10.14309/ajg.0000000000000051

Source DB:  PubMed          Journal:  Am J Gastroenterol        ISSN: 0002-9270            Impact factor:   10.864


  10 in total

1.  Inactivation of mesotrypsin by chymotrypsin C prevents trypsin inhibitor degradation.

Authors:  Vanda Toldi; András Szabó; Miklós Sahin-Tóth
Journal:  J Biol Chem       Date:  2020-02-03       Impact factor: 5.157

2.  Single nucleotide polymorphisms in CEL-HYB1 increase risk for chronic pancreatitis through proteotoxic misfolding.

Authors:  Brett M Cassidy; Sammy Zino; Karianne Fjeld; Anders Molven; Mark E Lowe; Xunjun Xiao
Journal:  Hum Mutat       Date:  2020-09-09       Impact factor: 4.878

3.  Loss-of-function variant in chymotrypsin like elastase 3B (CELA3B) is associated with non-alcoholic chronic pancreatitis.

Authors:  Andrea Tóth; Alexandra Demcsák; Florence Zankl; Grzegorz Oracz; Lara Sophie Unger; Peter Bugert; Helmut Laumen; Andrea Párniczky; Péter Hegyi; Jonas Rosendahl; Tomasz Gambin; Rafał Płoski; Dorota Koziel; Stanisław Gluszek; Fredrik Lindgren; J Matthias Löhr; Miklós Sahin-Tóth; Heiko Witt; Agnieszka Magdalena Rygiel; Maren Ewers; Eszter Hegyi
Journal:  Pancreatology       Date:  2022-06-23       Impact factor: 3.977

4.  Arg236 in human chymotrypsin B2 (CTRB2) is a key determinant of high enzyme activity, trypsinogen degradation capacity, and protection against pancreatitis.

Authors:  Bálint Zoltán Németh; Alexandra Demcsák; András Micsonai; Bence Kiss; Gitta Schlosser; Andrea Geisz; Eszter Hegyi; Miklós Sahin-Tóth; Gábor Pál
Journal:  Biochim Biophys Acta Proteins Proteom       Date:  2022-08-05       Impact factor: 4.125

5.  Cigarette smoke toxin hydroquinone and misfolding pancreatic lipase variant cooperatively promote endoplasmic reticulum stress and cell death.

Authors:  Norbert Kassay; Vanda Toldi; József Tőzsér; András Szabó
Journal:  PLoS One       Date:  2022-06-15       Impact factor: 3.752

6.  Bicarbonate defective CFTR variants increase risk for chronic pancreatitis: A meta-analysis.

Authors:  Gergő Berke; Noémi Gede; Letícia Szadai; Klementina Ocskay; Péter Hegyi; Miklós Sahin-Tóth; Eszter Hegyi
Journal:  PLoS One       Date:  2022-10-20       Impact factor: 3.752

7.  Impact of genetic testing and smoking on the distribution of risk factors in patients with recurrent acute and chronic pancreatitis.

Authors:  Merve Gurakar; Niloofar Y Jalaly; Mahya Faghih; Tina Boortalary; Javad R Azadi; Mouen A Khashab; Christopher Fan; Anthony N Kalloo; Atif Zaheer; Vikesh K Singh; Elham Afghani
Journal:  Scand J Gastroenterol       Date:  2021-10-18       Impact factor: 3.027

Review 8.  Acute Pancreatitis: Genetic Risk and Clinical Implications.

Authors:  Frank U Weiss; Felix Laemmerhirt; Markus M Lerch
Journal:  J Clin Med       Date:  2021-01-07       Impact factor: 4.241

9.  Misfolding-induced chronic pancreatitis in CPA1 N256K mutant mice is unaffected by global deletion of Ddit3/Chop.

Authors:  Balázs Csaba Németh; Alexandra Demcsák; Andrea Geisz; Miklós Sahin-Tóth
Journal:  Sci Rep       Date:  2022-04-15       Impact factor: 4.996

10.  Scale and Scope of Gene-Alcohol Interactions in Chronic Pancreatitis: A Systematic Review.

Authors:  Jian-Min Chen; Anthony F Herzig; Emmanuelle Génin; Emmanuelle Masson; David N Cooper; Claude Férec
Journal:  Genes (Basel)       Date:  2021-03-25       Impact factor: 4.096

  10 in total

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