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Literature DB >> 30738127

Granulocyte death mediated by specific antibodies in intravenous immunoglobulin (IVIG).

Stefanie Graeter¹, Hans-Uwe Simon², Stephan von Gunten³.

Abstract

Granulocytes are key effector cells of the innate immune system that cause substantial by-stander tissue damage in a broad range of inflammatory disorders. Specific antibodies with the potential to regulate granulocyte survival are present in polyclonal intravenous immunoglobulin (IVIG) preparations and may contribute to the anti-inflammatory effects of high-dose IVIG therapy. Here, we discuss idiosyncratic characteristics of antibodies to FAS and Siglecs contained in IVIG pertaining to granulocyte death regulation and highlight implications for research and clinical practice.

Entities: Disease

Keywords: Eosinophils; FAS; Granulocytes; High-dose IVIG; IVIG; Immunomodulation; Intravenous immunoglobulin; Neutrophils; Siglec-8; Siglec-9

Year: 2019 PMID： 30738127 DOI： 10.1016/j.phrs.2019.02.007

Source DB: PubMed Journal: Pharmacol Res ISSN： 1043-6618 Impact factor: 7.658

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Cited

10 in total

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2. Enhanced Pro-apoptotic Effects of Fe(II)-Modified IVIG on Human Neutrophils.

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3. Immunoglobulin Therapy in a Patient With Severe Chikungunya Fever and Vesiculobullous Lesions.

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Review 7. High-Dose Intravenous Immunoglobulins in the Treatment of Severe Acute Viral Pneumonia: The Known Mechanisms and Clinical Effects.

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