Literature DB >> 30637644

Hierarchical assessment of host factors influencing the spontaneous resolution of hepatitis C infection.

Paola Jocelan Scarin Provazzi1, Livia Maria Gonçalves Rossi1, Bruno Moreira Carneiro1, Valeria Chamas Miura1, Plinio Cesar Rodrigues Rosa1, Lucas Rodrigues de Carvalho1, Stephane Tereza Queiroz de Andrade1, Roberta Maria Fachini2, Rejane Maria Tommasini Grotto3, Giovanni Faria Silva3, Carlos Roberto Valêncio4, Paulo Scarpelini Neto4, José Antonio Cordeiro4, Mauricio Lacerda Nogueira5, Paula Rahal6.   

Abstract

BACKGROUND: Hepatitis C virus (HCV) infection is associated with chronic liver disease, resulting in cirrhosis and hepatocellular carcinoma. Approximately 20% of HCV infections are spontaneously resolved. Here, we assessed the hierarchical relevance of host factors contributing to viral clearance.
METHODS: DNA samples from 40 resolved infections and 40 chronic HCV patients paired by age were analyzed. Bivariate analysis was performed to rank the importance of each contributing factor in spontaneous HCV clearance.
RESULTS: Interestingly, 63.6% of patients with resolved infections exhibited the protective genotype CC for SNP rs12979860. Additionally, 59.3% of patients with resolved infections displayed the protective genotype TT/TT for SNP ss469415590. Moreover, a ranking of clearance factors was estimated. In order of importance, the IL28B CC genotype (OR 0.197, 95% CI 0.072-0.541) followed by the INFL4 TT/TT genotype (OR 0.237, 95% CI 0.083-0.679), and female gender (OR 0.394, 95% CI 0.159-0.977) were the main predictors for clearance of HCV infection.
CONCLUSIONS: HCV clearance is multifactorial and the contributing factors display a hierarchical order. Identifying all elements playing role in HCV clearance is of the most importance for HCV-related disease management. Dissecting the relevance of each contributing factor will certainly improve our understanding of the pathogenesis of HCV infection.

Entities:  

Keywords:  HCV; Hepatitis C; IL28B; MAVS; SNP rs12979860

Mesh:

Substances:

Year:  2018        PMID: 30637644      PMCID: PMC6863435          DOI: 10.1007/s42770-018-0008-3

Source DB:  PubMed          Journal:  Braz J Microbiol        ISSN: 1517-8382            Impact factor:   2.476


  63 in total

1.  Hepatic ISG expression is associated with genetic variation in interleukin 28B and the outcome of IFN therapy for chronic hepatitis C.

Authors:  Masao Honda; Akito Sakai; Tatsuya Yamashita; Yasunari Nakamoto; Eishiro Mizukoshi; Yoshio Sakai; Taro Yamashita; Mikiko Nakamura; Takayoshi Shirasaki; Katsuhisa Horimoto; Yasuhito Tanaka; Katsushi Tokunaga; Masashi Mizokami; Shuichi Kaneko
Journal:  Gastroenterology       Date:  2010-04-29       Impact factor: 22.682

2.  IFNL4 ss469415590 variant is a better predictor than rs12979860 of pegylated interferon-alpha/ribavirin therapy failure in hepatitis C virus/HIV-1 coinfected patients.

Authors:  Sandra Franco; Ester Aparicio; Mariona Parera; Bonaventura Clotet; Cristina Tural; Miguel Angel Martinez
Journal:  AIDS       Date:  2014-01-02       Impact factor: 4.177

3.  Sexual transmission of hepatitis C virus.

Authors:  F M Tengan; J Eluf-Neto; N P Cavalheiro ; A A Barone
Journal:  Rev Inst Med Trop Sao Paulo       Date:  2001 May-Jun       Impact factor: 1.846

4.  Human papillomavirus (HPV) study of 2916 cytological samples by PCR and DNA sequencing: genotype spectrum of patients from the west German area.

Authors:  Norbert Speich; Christoph Schmitt; Reinhard Bollmann; Magdolna Bollmann
Journal:  J Med Microbiol       Date:  2004-02       Impact factor: 2.472

5.  Acute hepatitis C in HIV-infected men who have sex with men.

Authors:  J Ghosn; S Pierre-François; V Thibault; C Duvivier; R Tubiana; A Simon; M A Valantin; S Dominguez; E Caumes; C Katlama
Journal:  HIV Med       Date:  2004-07       Impact factor: 3.180

6.  Routine genotyping of human papillomavirus samples in Denmark.

Authors:  Thomas Johnson; Karin Bryder; Sylvie Corbet; Anders Fomsgaard
Journal:  APMIS       Date:  2003-03       Impact factor: 3.205

7.  Cleavage of mitochondrial antiviral signaling protein in the liver of patients with chronic hepatitis C correlates with a reduced activation of the endogenous interferon system.

Authors:  Pantxika Bellecave; Magdalena Sarasin-Filipowicz; Olivier Donzé; Audrey Kennel; Jérôme Gouttenoire; Etienne Meylan; Luigi Terracciano; Jürg Tschopp; Christoph Sarrazin; Thomas Berg; Darius Moradpour; Markus H Heim
Journal:  Hepatology       Date:  2010-04       Impact factor: 17.425

8.  IL28B is associated with response to chronic hepatitis C interferon-alpha and ribavirin therapy.

Authors:  Vijayaprakash Suppiah; Max Moldovan; Golo Ahlenstiel; Thomas Berg; Martin Weltman; Maria Lorena Abate; Margaret Bassendine; Ulrich Spengler; Gregory J Dore; Elizabeth Powell; Stephen Riordan; David Sheridan; Antonina Smedile; Vincenzo Fragomeli; Tobias Müller; Melanie Bahlo; Graeme J Stewart; David R Booth; Jacob George
Journal:  Nat Genet       Date:  2009-09-13       Impact factor: 38.330

9.  IFNL4 ss469415590 polymorphism contributes to treatment decisions in patients with chronic hepatitis C virus genotype 1b, but not 2a, infection.

Authors:  Ruihong Wu; Xiumei Chi; Xiaomei Wang; Haibo Sun; Juan Lv; Xiuzhu Gao; Ge Yu; Fei Kong; Hongqin Xu; Rui Hua; Jing Jiang; Bing Sun; Jin Zhong; Yu Pan; Junqi Niu
Journal:  Infect Genet Evol       Date:  2016-01-26       Impact factor: 3.342

10.  Hepatitis C virus NS3-4A inhibits the peroxisomal MAVS-dependent antiviral signalling response.

Authors:  Ana R Ferreira; Ana C Magalhães; Fátima Camões; Ana Gouveia; Marta Vieira; Jonathan C Kagan; Daniela Ribeiro
Journal:  J Cell Mol Med       Date:  2016-02-10       Impact factor: 5.310

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