Literature DB >> 30620398

Systemic proinflammation after Mycobacterium tuberculosis infection was correlated to the gut microbiome in HIV-uninfected humans.

Shiang-Fen Huang1,2,3, Ying-Ying Yang1,3,4, Kun-Ta Chou5,6, Chang-Phone Fung1,2, Fu-Der Wang2,5, Wei-Juin Su5,6.   

Abstract

BACKGROUND: The dysbiosis of gut microbiome and interaction with host immunity after Mycobacterium tuberculosis (MTB) infection are under investigation. We had found fatigue symptom concurrent with dysbiosis by decreasing the ratio of Firmicutes to Bacteroidetes (F/B ratio) in active tuberculosis (TB). The study aims to assess the inflammatory biomarkers and their interaction with gut microbiome in active TB and latent TB infection before starting anti-TB regimens. MATERIALS AND
METHOD: Interleukin-1 beta (IL-1B), IL-4, IL-6, IL-10, CD3+, CD4+, CD8+ T cells and interferon-gamma (IFN-γ) releasing assay (IGRA) were measured in 25 active TB patients, 32 LTBI subjects and 23 healthy controls (HC). Gut microbiome profiles were obtained using 16S rRNA MiSeq sequencing method.
RESULTS: The leucocytosis (7032 ± 387 cell/cum, P < 0.05), increase in IL-6 (229.7 ± 104 µg/dL, P < 0.05), and decrease in IL-4 (0.27 µg/dL ± 0.1, P < 0.05) were presented in active TB. The proportion of polymorphic neutrophil (PMN) in peripheral blood was positively related to the relative abundance of Bacteroidetes in LTBI and active TB (R2  = 0.23, P < 0.05). The F/B ratio was positively related to the detectable IL-1B in TB (R2  = 0.97, P < 0.01) and to the IL-4 in LTBI (R2  = 0.27, P < 0.05). In LTBI, the relative abundances of Coriobacteriaceae were positively related to the secretion of IFN-gamma against MTB-antigens more likely associated with of CD4+ T cell (R2  = 0.42, P < 0.05).
CONCLUSION: In active TB, dysbiosis with higher relative abundances of Bacteroidetes in stool and low F/B ratio was related to systemic proinflammation. In LTBI, dose-response relationship between peripheral PMN and relative abundances of Bacteroidetes was remained but not leads to systemic inflammation.
© 2019 Stichting European Society for Clinical Investigation Journal Foundation.

Entities:  

Keywords:  16S rRNA gene; gut microbiome; proinflammation; tuberculosis

Year:  2019        PMID: 30620398     DOI: 10.1111/eci.13068

Source DB:  PubMed          Journal:  Eur J Clin Invest        ISSN: 0014-2972            Impact factor:   4.686


  3 in total

Review 1.  The gut microbiome in tuberculosis susceptibility and treatment response: guilty or not guilty?

Authors:  Osagie A Eribo; Nelita du Plessis; Mumin Ozturk; Reto Guler; Gerhard Walzl; Novel N Chegou
Journal:  Cell Mol Life Sci       Date:  2019-11-15       Impact factor: 9.261

Review 2.  Microbiome-immune interactions in tuberculosis.

Authors:  Giorgia Mori; Mark Morrison; Antje Blumenthal
Journal:  PLoS Pathog       Date:  2021-04-15       Impact factor: 6.823

3.  Alterations in the Gut Microbiome of Individuals With Tuberculosis of Different Disease States.

Authors:  Yue Wang; Yali Deng; Nianqiang Liu; Yanggui Chen; Yuandong Jiang; Zihao Teng; Zhi Ma; Yuxue Chang; Yang Xiang
Journal:  Front Cell Infect Microbiol       Date:  2022-03-29       Impact factor: 5.293

  3 in total

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