Literature DB >> 30603363

Factors involved in decreasing the therapeutic effect of sitagliptin: a subanalysis of the JAMP study.

Hideo Nunome1, Hiroshi Sakura2, Naotake Hashimoto3, Kazuo Sasamoto4, Hiroshi Ohashi5, Sumiko Hasumi6, Noriko Ujihara7, Tadasu Kasahara8, Osamu Tomonaga9, Masashi Honda10, Yasuhiko Iwamoto11.   

Abstract

OBJECTIVE: As a subanalysis of the Januvia Multicenter Prospective Trial in Type 2 Diabetes (JAMP study), we examined factors that decreased blood glucose control effect of sitagliptin after 3 months and patients requiring an addition or increase of diabetes treatment.
METHODS: We selected patients in whom glycated hemoglobin (HbA1c) levels decreased by month 3 after initiation of sitagliptin treatment and conducted two analyses: (1) in patients who did not change drugs until month 12, we compared changes in HbA1c levels between concomitant drugs and examined factors that decreased blood glucose control effect of sitagliptin; (2) compared changes in HbA1c levels and backgrounds between patients who did and did not require an addition to or increased dose of the antidiabetic agent.
RESULTS: Four hundred and ninety-eight patients were chosen. In 369 patients without drug change until month 12, changes in HbA1c levels during months 3-12 were not significantly different among concomitant drugs; factors causing rebound HbA1c were smoking and weight gain. Patient characteristics were compared between those who did and did not require an additional drug or a dose increase (n = 114) (n = 384). Drug changes were associated with longer disease duration, younger age, higher rate of smoking, and higher degree of insulin resistance but not with concomitantly administered drugs.
CONCLUSION: Smoking and weight gain were factors that decreased the effect of sitagliptin on reducing blood glucose levels. Differences in concomitant drugs did not affect sitagliptin's effects on glycemic control. A dose increase or the addition of the antidiabetic drug was not associated with concomitant drugs.

Entities:  

Keywords:  DPP4-inhibitor; Decreasing the therapeutic effect; HbA1c rebound factor; Sitagliptin; Type 2 diabetes mellitus

Year:  2017        PMID: 30603363      PMCID: PMC6224911          DOI: 10.1007/s13340-017-0340-0

Source DB:  PubMed          Journal:  Diabetol Int        ISSN: 2190-1678


  22 in total

1.  Efficacy and safety of sitagliptin monotherapy in Japanese patients with type 2 diabetes.

Authors:  Kenji Nonaka; Taro Kakikawa; Asako Sato; Kotoba Okuyama; Go Fujimoto; Naoki Kato; Hideyo Suzuki; Yukio Hirayama; Tuli Ahmed; Michael J Davies; Peter P Stein
Journal:  Diabetes Res Clin Pract       Date:  2007-10-22       Impact factor: 5.602

2.  Preprandial single oral dose of sitagliptin does not affect circulating ghrelin and gastrin levels in normal subjects.

Authors:  Chen-Ling Huang; Chung-Huei Hsu; Kuo-Cherh Huang; Hsiu-Yueh Su; Shuen-Fu Weng
Journal:  Pharmacology       Date:  2010-02-09       Impact factor: 2.547

3.  Chronic inhibition of dipeptidyl peptidase-4 with a sitagliptin analog preserves pancreatic beta-cell mass and function in a rodent model of type 2 diabetes.

Authors:  James Mu; John Woods; Yun-Ping Zhou; Ranabir Sinha Roy; Zhihua Li; Emanuel Zycband; Yue Feng; Lan Zhu; Cai Li; Andrew D Howard; David E Moller; Nancy A Thornberry; Bei B Zhang
Journal:  Diabetes       Date:  2006-06       Impact factor: 9.461

4.  Reduced beta-cell mass and expression of oxidative stress-related DNA damage in the islet of Japanese Type II diabetic patients.

Authors:  H Sakuraba; H Mizukami; N Yagihashi; R Wada; C Hanyu; S Yagihashi
Journal:  Diabetologia       Date:  2002-01       Impact factor: 10.122

5.  Efficacy and safety of sitagliptin monotherapy compared with voglibose in Japanese patients with type 2 diabetes: a randomized, double-blind trial.

Authors:  Y Iwamoto; N Tajima; T Kadowaki; K Nonaka; T Taniguchi; M Nishii; J C Arjona Ferreira; J M Amatruda
Journal:  Diabetes Obes Metab       Date:  2010-07       Impact factor: 6.577

6.  Glycemic durability of rosiglitazone, metformin, or glyburide monotherapy.

Authors:  Steven E Kahn; Steven M Haffner; Mark A Heise; William H Herman; Rury R Holman; Nigel P Jones; Barbara G Kravitz; John M Lachin; M Colleen O'Neill; Bernard Zinman; Giancarlo Viberti
Journal:  N Engl J Med       Date:  2006-12-04       Impact factor: 91.245

7.  Effect of sitagliptin monotherapy on serum total ghrelin levels in people with type 2 diabetes.

Authors:  Özen Öz; Sinem Kıyıcı; Canan Ersoy; Soner Cander; Hakan Yorulmaz; Cuma Bülent Gül; Oğuz Kaan Ünal; Emre Sarandol; Emine Kırhan; Deniz Sığırlı; Erdinç Ertürk; Ercan Tuncel; Sazi Imamoğlu
Journal:  Diabetes Res Clin Pract       Date:  2011-08-19       Impact factor: 5.602

8.  Chronic administration of alogliptin, a novel, potent, and highly selective dipeptidyl peptidase-4 inhibitor, improves glycemic control and beta-cell function in obese diabetic ob/ob mice.

Authors:  Yusuke Moritoh; Koji Takeuchi; Tomoko Asakawa; Osamu Kataoka; Hiroyuki Odaka
Journal:  Eur J Pharmacol       Date:  2008-04-10       Impact factor: 4.432

9.  Beta-cell deficit and increased beta-cell apoptosis in humans with type 2 diabetes.

Authors:  Alexandra E Butler; Juliette Janson; Susan Bonner-Weir; Robert Ritzel; Robert A Rizza; Peter C Butler
Journal:  Diabetes       Date:  2003-01       Impact factor: 9.461

10.  Dipeptidyl peptidase 4 is a novel adipokine potentially linking obesity to the metabolic syndrome.

Authors:  Daniela Lamers; Susanne Famulla; Nina Wronkowitz; Sonja Hartwig; Stefan Lehr; D Margriet Ouwens; Kristin Eckardt; Jean M Kaufman; Mikael Ryden; Stefan Müller; Franz-Georg Hanisch; Johannes Ruige; Peter Arner; Henrike Sell; Juergen Eckel
Journal:  Diabetes       Date:  2011-05-18       Impact factor: 9.461

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