Literature DB >> 30579849

Epigenome-wide meta-analysis of DNA methylation and childhood asthma.

Sarah E Reese1, Cheng-Jian Xu2, Herman T den Dekker3, Mi Kyeong Lee1, Sinjini Sikdar1, Carlos Ruiz-Arenas4, Simon K Merid5, Faisal I Rezwan6, Christian M Page7, Vilhelmina Ullemar8, Phillip E Melton9, Sam S Oh10, Ivana V Yang11, Kimberley Burrows12, Cilla Söderhäll13, Dereje D Jima14, Lu Gao15, Ryan Arathimos16, Leanne K Küpers17, Matthias Wielscher18, Peter Rzehak19, Jari Lahti20, Catherine Laprise21, Anne-Marie Madore22, James Ward1, Brian D Bennett1, Tianyuan Wang1, Douglas A Bell1, Judith M Vonk23, Siri E Håberg24, Shanshan Zhao1, Robert Karlsson8, Elysia Hollams25, Donglei Hu10, Adam J Richards11, Anna Bergström26, Gemma C Sharp27, Janine F Felix28, Mariona Bustamante29, Olena Gruzieva26, Rachel L Maguire30, Frank Gilliland15, Nour Baïz31, Ellen A Nohr32, Eva Corpeleijn33, Sylvain Sebert34, Wilfried Karmaus35, Veit Grote19, Eero Kajantie36, Maria C Magnus37, Anne K Örtqvist8, Celeste Eng10, Andrew H Liu38, Inger Kull39, Vincent W V Jaddoe28, Jordi Sunyer40, Juha Kere41, Cathrine Hoyo42, Isabella Annesi-Maesano31, Syed Hasan Arshad43, Berthold Koletzko19, Bert Brunekreef44, Elisabeth B Binder45, Katri Räikkönen46, Eva Reischl47, John W Holloway48, Marjo-Riitta Jarvelin49, Harold Snieder33, Nabila Kazmi16, Carrie V Breton15, Susan K Murphy50, Göran Pershagen26, Josep Maria Anto40, Caroline L Relton12, David A Schwartz11, Esteban G Burchard51, Rae-Chi Huang25, Wenche Nystad24, Catarina Almqvist52, A John Henderson53, Erik Melén54, Liesbeth Duijts55, Gerard H Koppelman56, Stephanie J London57.   

Abstract

BACKGROUND: Epigenetic mechanisms, including methylation, can contribute to childhood asthma. Identifying DNA methylation profiles in asthmatic patients can inform disease pathogenesis.
OBJECTIVE: We sought to identify differential DNA methylation in newborns and children related to childhood asthma.
METHODS: Within the Pregnancy And Childhood Epigenetics consortium, we performed epigenome-wide meta-analyses of school-age asthma in relation to CpG methylation (Illumina450K) in blood measured either in newborns, in prospective analyses, or cross-sectionally in school-aged children. We also identified differentially methylated regions.
RESULTS: In newborns (8 cohorts, 668 cases), 9 CpGs (and 35 regions) were differentially methylated (epigenome-wide significance, false discovery rate < 0.05) in relation to asthma development. In a cross-sectional meta-analysis of asthma and methylation in children (9 cohorts, 631 cases), we identified 179 CpGs (false discovery rate < 0.05) and 36 differentially methylated regions. In replication studies of methylation in other tissues, most of the 179 CpGs discovered in blood replicated, despite smaller sample sizes, in studies of nasal respiratory epithelium or eosinophils. Pathway analyses highlighted enrichment for asthma-relevant immune processes and overlap in pathways enriched both in newborns and children. Gene expression correlated with methylation at most loci. Functional annotation supports a regulatory effect on gene expression at many asthma-associated CpGs. Several implicated genes are targets for approved or experimental drugs, including IL5RA and KCNH2.
CONCLUSION: Novel loci differentially methylated in newborns represent potential biomarkers of risk of asthma by school age. Cross-sectional associations in children can reflect both risk for and effects of disease. Asthma-related differential methylation in blood in children was substantially replicated in eosinophils and respiratory epithelium. Published by Elsevier Inc.

Entities:  

Keywords:  Epigenetics; asthma; childhood; drug development; methylation; newborn

Mesh:

Substances:

Year:  2018        PMID: 30579849      PMCID: PMC6556405          DOI: 10.1016/j.jaci.2018.11.043

Source DB:  PubMed          Journal:  J Allergy Clin Immunol        ISSN: 0091-6749            Impact factor:   10.793


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