Literature DB >> 30579806

Efficacy of alirocumab for reducing plaque vulnerability: Study protocol for ALTAIR, a randomized controlled trial in Japanese patients with coronary artery disease receiving rosuvastatin.

Hiromasa Otake1, Yoichiro Sugizaki2, Takayoshi Toba2, Yuichiro Nagano2, Yoshiro Tsukiyama2, Ken-Ichi Yanaka2, Hiroyuki Yamamoto2, Akira Nagasawa2, Hiroyuki Onishi2, Ryo Takeshige2, Shinsuke Nakano2, Yoichiro Matsuoka2, Kosuke Tanimura2, Hiroyuki Kawamori2, Toshiro Shinke2, Ken-Ichi Hirata2.   

Abstract

BACKGROUND: Although a recent clinical trial demonstrated that alirocumab, a proprotein convertase subtilisin/kexin type 9 (PCSK9) inhibitor, significantly reduces the incidence of acute coronary events, the impact of alirocumab on plaque stabilization remains uncertain. The Efficacy of ALirocumab for Thin-cap fibroatheroma in patients with coronary Artery disease estImated by optical coherence tomogRaphy (ALTAIR) study will investigate the effect of alirocumab on thin-cap fibroatheroma (TCFA) in Japanese patients who underwent recent percutaneous coronary intervention (PCI). METHODS AND
DESIGN: ALTAIR is a phase IV, open-label, randomized, parallel-group, single-center study involving blinded optical coherence tomography (OCT) image analysis in Japanese adults hospitalized for PCI and having suboptimal control of low-density lipoprotein cholesterol (LDL-C) levels (>70mg/dL) despite statin therapy. Patients will be randomized (1:1) to the alirocumab arm (alirocumab 75mg every 2 weeks added to rosuvastatin 10mg/day) or the standard-of-care arm (rosuvastatin 10mg/day, with initiation and/or dose adjustment of non-statin lipid-lowering to achieve an LDL-C target of <70mg/dL). OCT imaging will be conducted at baseline and at week 36 (post-treatment). The primary objective is to compare the alirocumab and standard-of-care arms regarding the change in TCFA fibrous-cap thickness after 9 months of treatment.
CONCLUSION: The outcomes of ALTAIR (ClinicalTrials.gov identifier: NCT03552432) will provide insights into the effect of alirocumab on plaque vulnerability following PCI in patients with suboptimal LDL-C control despite stable statin therapy.
Copyright © 2018 Japanese College of Cardiology. Published by Elsevier Ltd. All rights reserved.

Entities:  

Keywords:  Alirocumab; Coronary artery disease; Percutaneous coronary intervention; Rosuvastatin

Mesh:

Substances:

Year:  2018        PMID: 30579806     DOI: 10.1016/j.jjcc.2018.11.012

Source DB:  PubMed          Journal:  J Cardiol        ISSN: 0914-5087            Impact factor:   3.159


  4 in total

1.  PCSK9 monoclonal antibodies for the primary and secondary prevention of cardiovascular disease.

Authors:  Amand F Schmidt; John-Paul L Carter; Lucy S Pearce; John T Wilkins; John P Overington; Aroon D Hingorani; J P Casas
Journal:  Cochrane Database Syst Rev       Date:  2020-10-20

2.  Effect of alirocumab on coronary plaque in patients with coronary artery disease assessed by optical coherence tomography.

Authors:  Fei Gao; Zhi Jian Wang; Xiao Teng Ma; Hua Shen; Li Xia Yang; Yu Jie Zhou
Journal:  Lipids Health Dis       Date:  2021-09-12       Impact factor: 3.876

3.  Impact of PCSK9 Inhibition on Proinflammatory Cytokines and Matrix Metalloproteinases Release in Patients with Mixed Hyperlipidemia and Vulnerable Atherosclerotic Plaque.

Authors:  Marcin Basiak; Michal Kosowski; Marcin Hachula; Boguslaw Okopien
Journal:  Pharmaceuticals (Basel)       Date:  2022-06-27

4.  Impact of Alirocumab on Release Markers of Atherosclerotic Plaque Vulnerability in Patients with Mixed Hyperlipidemia and Vulnerable Atherosclerotic Plaque.

Authors:  Michał Kosowski; Marcin Basiak; Marcin Hachuła; Bogusław Okopień
Journal:  Medicina (Kaunas)       Date:  2022-07-21       Impact factor: 2.948

  4 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.