Literature DB >> 3050797

Non-traumatic ischaemic myelopathy: a review of 25 cases.

S W Kim1, R C Kim, B H Choi, S K Gordon.   

Abstract

The causes of ischaemic myelopathy are described in 25 patients. Nine developed following surgical manipulation or traumatic laceration of the aorta, 1 following intercostal artery ligation, 3 following aortic aneurysm dissection, 2 following myocardial infarction and/or cardiac arrest, 7 in the absence of any specifically identifiable predisposing factors, and 3 in association with decompression sickness. The degree of clinical recovery was greater among those with incomplete spastic (as opposed to complete flaccid) paralysis and among those in whom sensory loss below the level of injury was incomplete. Despite the diversity of mechanisms that may lead to the development of spinal cord ischaemia, structural damage seems in most instances to affect either grey matter or white matter predominantly. Some of the possible reasons for these preferential sites of damage are discussed.

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Year:  1988        PMID: 3050797     DOI: 10.1038/sc.1988.40

Source DB:  PubMed          Journal:  Paraplegia        ISSN: 0031-1758


  9 in total

1.  Development of AMPA receptor and GABA B receptor-sensitive spinal hyper-reflexia after spinal air embolism in rat: a systematic neurological, electrophysiological and qualitative histopathological study.

Authors:  Osamu Kakinohana; Miriam Scadeng; Jose A Corleto; Juraj Sevc; Nadezda Lukacova; Martin Marsala
Journal:  Exp Neurol       Date:  2012-06-18       Impact factor: 5.330

2.  Late deterioration after decompression illness affecting the spinal cord.

Authors:  J Dyer; P Millac
Journal:  Br J Sports Med       Date:  1996-12       Impact factor: 13.800

3.  Recovery after spinal cord infarcts: long-term outcome in 115 patients.

Authors:  Carrie E Robertson; Robert D Brown; Eelco F M Wijdicks; Alejandro A Rabinstein
Journal:  Neurology       Date:  2011-12-28       Impact factor: 9.910

4.  Peripheral spinal cord hypointensity on T2-weighted MR images: a reliable imaging sign of venous hypertensive myelopathy.

Authors:  R W Hurst; R I Grossman
Journal:  AJNR Am J Neuroradiol       Date:  2000-04       Impact factor: 3.825

Review 5.  Spinal Cord Infarcts: Risk Factors, Management, and Prognosis.

Authors:  Deena M Nasr; Alejandro Rabinstein
Journal:  Curr Treat Options Neurol       Date:  2017-08       Impact factor: 3.598

6.  Prognosis and recovery in ischaemic and traumatic spinal cord injury: clinical and electrophysiological evaluation.

Authors:  E Iseli; A Cavigelli; V Dietz; A Curt
Journal:  J Neurol Neurosurg Psychiatry       Date:  1999-11       Impact factor: 10.154

7.  Aortic pathology revealed by MRI in patients with clinical suspicion of spinal disease.

Authors:  E M Larsson; M Heiling; S Holtås
Journal:  Neuroradiology       Date:  1993       Impact factor: 2.804

Review 8.  Acute aortic thrombosis associated with spinal cord infarction in nephrotic syndrome.

Authors:  Y Fujigaki; M Kimura; T Shimizu; N Ikegaya; T Arai; A Hishida; E Kaneko; T Hachiya
Journal:  Clin Investig       Date:  1992-07

9.  "Venous congestion" as a cause of subcortical white matter T2 hypointensity on magnetic resonance images.

Authors:  Jayaprakash Harsha Kamble; Krishnan Parameswaran
Journal:  Ann Indian Acad Neurol       Date:  2016 Jul-Sep       Impact factor: 1.383

  9 in total

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