Literature DB >> 30456866

Liraglutide pharmacotherapy reduces body weight and improves glycaemic control in juvenile obese/hyperglycaemic male and female rats.

Claudia G Liberini1, Rinzin Lhamo1, Misgana Ghidewon1, Tyler Ling1, Nina Juntereal1, Jack Chen1, Anh Cao1, Lauren M Stein1, Matthew R Hayes1.   

Abstract

AIMS: To examine whether the glucagon-like peptide-1 receptor agonist liraglutide could be used in juvenile male and female rats as an anti-obesity/diabetic pharmaceutical to prevent not only adolescent obesity/hyperglycaemia, but also early-adult onset obesity.
MATERIAL AND METHODS: Pregnant dams were fed either standard chow or a high-fat, high-sucrose diet (HFSD) from gestational day 2, throughout pregnancy and lactation. Offspring were weaned onto the respective maternal diet. Juveniles received daily subcutaneous injection of liraglutide (50 μg/kg, from postnatal day [PND]30 to PND40 and 200 μg/kg from PND40 to PND60) or vehicle. Food intake, body weight and glycaemic levels were evaluated across the experimental period.
RESULTS: Chronic liraglutide administration in juveniles prevented body weight gain in males and retained a normoglycaemic profile in both male and female rats.
CONCLUSION: These preclinical data suggest that maternal and early-life consumption of an HFSD increases caloric intake, body weight gain and hyperglycaemia, a collective set of unwanted metabolic effects that appear to be treatable in juveniles with liraglutide pharmacotherapy intervention.
© 2018 John Wiley & Sons Ltd.

Entities:  

Keywords:  GLP-1 analogue; animal pharmacology; liraglutide; neuropharmacology

Mesh:

Substances:

Year:  2018        PMID: 30456866      PMCID: PMC7274726          DOI: 10.1111/dom.13591

Source DB:  PubMed          Journal:  Diabetes Obes Metab        ISSN: 1462-8902            Impact factor:   6.577


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1.  Early life overnutrition impairs plasticity of non-neuronal brainstem cells and drives obesity in offspring across development in rats.

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