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Literature DB >> 30448718

Treatment of intractable resting tremor of spinocerebellar ataxia 42 with zonisamide.

Naoyuki Hara¹, Tomohisa Nezu², Keitaro Kobatake³, Hiroyuki Morino⁴, Hideshi Kawakami⁴, Hirofumi Maruyama⁵.

Abstract

Entities: Chemical Disease

Keywords: CACNA1G; Spinocerebellar ataxia; T-type calcium channel; Tremor; Zonisamide

Mesh：

Substances：

Year: 2018 PMID： 30448718 DOI： 10.1016/j.jns.2018.11.013

Source DB: PubMed Journal: J Neurol Sci ISSN： 0022-510X Impact factor: 3.181

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1 in total

1. Zonisamide can ameliorate the voltage-dependence alteration of the T-type calcium channel Ca_V3.1 caused by a mutation responsible for spinocerebellar ataxia.

Authors: Naoyuki Hara; Hiroyuki Morino; Yukiko Matsuda; Kenichi Satoh; Kouichi Hashimoto; Hirofumi Maruyama; Hideshi Kawakami
Journal: Mol Brain Date: 2020-11-26 Impact factor: 4.041

1 in total

Treatment of intractable resting tremor of spinocerebellar ataxia 42 with zonisamide.

1. Zonisamide can ameliorate the voltage-dependence alteration of the T-type calcium channel CaV3.1 caused by a mutation responsible for spinocerebellar ataxia.

1. Zonisamide can ameliorate the voltage-dependence alteration of the T-type calcium channel Ca_V3.1 caused by a mutation responsible for spinocerebellar ataxia.