Literature DB >> 30399106

Terminal Complement Activation in Preeclampsia.

Richard M Burwick1, Jesús A Velásquez, Catalina M Valencia, Jorge Gutiérrez-Marín, Francisco Edna-Estrada, Jaime L Silva, Juliana Trujillo-Otálvaro, Johanna Vargas-Rodríguez, Yamile Bernal, Alvaro Quintero, Mónica Rincón, Jorge E Tolosa.   

Abstract

OBJECTIVE: To evaluate whether C5b-9 concentrations in blood and urine are increased in preeclampsia with severe features.
METHODS: The Complement and Preeclampsia in the Americas study is a prospective, multicenter case-control study performed at six centers in Colombia from November 2015 to July 2016. The case group included women with preeclampsia with severe features, and the control group included women who were healthy or had chronic hypertension, gestational hypertension, or preeclampsia without severe features. We enrolled two women in the control group for every woman in the case group. Soluble C5b-9 concentrations were measured by enzyme-linked immunosorbent assays in blood and urine. The primary outcome was C5b-9 concentrations in women in the case group compared with all women in the control group, and the secondary outcome was C5b-9 levels in women in the case group compared with individual control subgroups. Differences were assessed by test of medians, and associations were further evaluated by receiver operating characteristic curve analysis and logistic regression with α=0.05.
RESULTS: Three hundred fifty-two patients were enrolled. Plasma C5b-9 concentrations did not differ significantly between women in the case group and those in the control group, but urine C5b-9 concentrations were higher in women in the case group (median [interquartile range] 9.9 [1.6-43.7] vs 1.8 [0.54-4.1] ng/mL, P<.001). In subgroup analysis, plasma C5b-9 concentrations were increased in women in the case group compared with healthy women in the control group (median [interquartile range] 2,778 [1,633-4,230] vs 1,374 [1,064-2,332] ng/mL, P<.001), and urine C5b-9 concentrations were increased in women in the case group compared with all control subgroups (P<.001). Using receiver operating characteristic analysis, urine C5b-9 concentrations differentiated preeclampsia with severe features from hypertensive women in the control group (area under the receiver operating characteristic curve 0.74, 95% CI 0.68-0.80). Urine C5b-9 22 ng/mL or greater (range 0-158.4 ng/mL) was the optimal cut point for diagnosis of preeclampsia with severe features with adjusted odds ratio of 10.0 (95% CI 3.5-28.8, P<.001).
CONCLUSION: Urinary excretion of terminal complement effector C5b-9 is higher in women with preeclampsia with severe features compared with women with other hypertensive disorders of pregnancy and women without hypertension.

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Year:  2018        PMID: 30399106     DOI: 10.1097/AOG.0000000000002980

Source DB:  PubMed          Journal:  Obstet Gynecol        ISSN: 0029-7844            Impact factor:   7.661


  10 in total

1.  Role of B1 and B2 lymphocytes in placental ischemia-induced hypertension.

Authors:  Connor F Laule; Evan J Odean; Cameron R Wing; Kate M Root; Kendra J Towner; Cassandra M Hamm; Jeffrey S Gilbert; Sherry D Fleming; Jean F Regal
Journal:  Am J Physiol Heart Circ Physiol       Date:  2019-08-09       Impact factor: 4.733

Review 2.  Review of the immune mechanisms of preeclampsia and the potential of immune modulating therapy.

Authors:  Ai-Ris Y Collier; Laura A Smith; S Ananth Karumanchi
Journal:  Hum Immunol       Date:  2021-02-05       Impact factor: 2.850

Review 3.  Pathogenesis of Preeclampsia and Therapeutic Approaches Targeting the Placenta.

Authors:  Manoj Kumar Jena; Neeta Raj Sharma; Matthew Petitt; Devika Maulik; Nihar Ranjan Nayak
Journal:  Biomolecules       Date:  2020-06-24

Review 4.  Preeclampsia: Maternal Systemic Vascular Disorder Caused by Generalized Endothelial Dysfunction Due to Placental Antiangiogenic Factors.

Authors:  Takuji Tomimatsu; Kazuya Mimura; Shinya Matsuzaki; Masayuki Endo; Keiichi Kumasawa; Tadashi Kimura
Journal:  Int J Mol Sci       Date:  2019-08-30       Impact factor: 5.923

Review 5.  Beyond Systemic Lupus Erythematosus and Anti-Phospholipid Syndrome: The Relevance of Complement From Pathogenesis to Pregnancy Outcome in Other Systemic Rheumatologic Diseases.

Authors:  Silvia Cavalli; Paola Adele Lonati; Maria Gerosa; Roberto Caporali; Rolando Cimaz; Cecilia Beatrice Chighizola
Journal:  Front Pharmacol       Date:  2022-02-15       Impact factor: 5.810

Review 6.  Molecular Advances in Preeclampsia and HELLP Syndrome.

Authors:  Angeliki Gardikioti; Theodora-Maria Venou; Eleni Gavriilaki; Evangelia Vetsiou; Ioulia Mavrikou; Konstantinos Dinas; Angelos Daniilidis; Efthymia Vlachaki
Journal:  Int J Mol Sci       Date:  2022-03-31       Impact factor: 5.923

7.  Common variants of fetal and maternal complement genes in preeclampsia: pregnancy specific complotype.

Authors:  Manu Banadakoppa; Meena Balakrishnan; Chandra Yallampalli
Journal:  Sci Rep       Date:  2020-03-16       Impact factor: 4.379

Review 8.  Mechanisms of Key Innate Immune Cells in Early- and Late-Onset Preeclampsia.

Authors:  Ingrid Aneman; Dillan Pienaar; Sonja Suvakov; Tatjana P Simic; Vesna D Garovic; Lana McClements
Journal:  Front Immunol       Date:  2020-08-18       Impact factor: 7.561

9.  Late first trimester circulating microparticle proteins predict the risk of preeclampsia < 35 weeks and suggest phenotypic differences among affected cases.

Authors:  Thomas F McElrath; David E Cantonwine; Kathryn J Gray; Hooman Mirzakhani; Robert C Doss; Najmuddin Khaja; Malik Khalid; Gail Page; Brian Brohman; Zhen Zhang; David Sarracino; Kevin P Rosenblatt
Journal:  Sci Rep       Date:  2020-10-21       Impact factor: 4.379

Review 10.  The Complement System, T Cell Response, and Cytokine Shift in Normotensive versus Pre-Eclamptic and Lupus Pregnancy.

Authors:  Eugen Ancuța; Radu Zamfir; Gabriel Martinescu; Dragoș Valentin Crauciuc; Codrina Ancuța
Journal:  J Clin Med       Date:  2021-12-07       Impact factor: 4.241

  10 in total

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