Literature DB >> 30371791

A Novel Somatostatin-Dopamine Chimera (BIM23B065) Reduced GH Secretion in a First-in-Human Clinical Trial.

Wadim M I de Boon1, Michiel J van Esdonk1,2, Frederik E Stuurman1,3, Nienke R Biermasz4, Laurent Pons5, Isabelle Paty5, Jacobus Burggraaf1,2.   

Abstract

Context: A somatostatin-dopamine chimera (BIM23B065) was under investigation to reduce GH secretion for the treatment of pituitary adenomas. Objective: To determine pharmacokinetics, safety, and tolerability and to monitor hormonal changes after single and multiple subcutaneous BIM23B065 administrations. Design: Randomized, double-blind, placebo-controlled, parallel-group design with five single and three 13-day multiple ascending-dose cohorts. Patients: A total of 63 healthy male white volunteers were enrolled (47 active, 16 placebo). Main Outcome Measures: Pharmacokinetics, GH, prolactin (PRL), IGF-1, GH after GHRH administration, and general clinical safety criteria.
Results: The maximum dosage of BIM23B065 administered in this study was 1.5 mg. BIM23B065 reduced the mean GH concentrations after 8 and 13 days of treatment. A decrease in GH release after GHRH administration indicated inhibition of the hypothalamic-pituitary-somatotropic axis. IGF-1 was not altered after single doses but showed a significant change from baseline after multiple dosing. PRL secretion was reduced in all subjects who were treated. Orthostatic hypotension and injection site reactions were commonly observed at high dosages. A 6-day uptitration period was included to successfully lower the cardiovascular effects in the multiple ascending dose part of the study. Conclusions: Proof of pharmacology of BIM23B065 was shown by a reduction in GH, IGF-1, and PRL concentrations in healthy male volunteers, supporting activity of the somatostatin analog and dopamine agonist moieties. The safety and tolerability of the higher dosing regions was limited mainly by orthostatic hypotension.

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Year:  2019        PMID: 30371791     DOI: 10.1210/jc.2018-01364

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  4 in total

Review 1.  Role of Somatostatin Signalling in Neuroendocrine Tumours.

Authors:  Olesja Rogoza; Kaspars Megnis; Marija Kudrjavceva; Aija Gerina-Berzina; Vita Rovite
Journal:  Int J Mol Sci       Date:  2022-01-27       Impact factor: 5.923

Review 2.  Current and Emerging Medical Therapies in Pituitary Tumors.

Authors:  Nicolas Sahakian; Frédéric Castinetti; Thierry Brue; Thomas Cuny
Journal:  J Clin Med       Date:  2022-02-12       Impact factor: 4.241

3.  Therapeutic Effect of a Novel Chimeric Molecule Targeting Both Somatostatin and Dopamine Receptors on Growth Hormone-Secreting Pituitary Adenomas.

Authors:  Jean Kim; Ju Hun Oh; Heather Harlem; Michael D Culler; Cheol Ryong Ku; Eun Jig Lee
Journal:  Endocrinol Metab (Seoul)       Date:  2020-03

4.  Quantification of the endogenous growth hormone and prolactin lowering effects of a somatostatin-dopamine chimera using population PK/PD modeling.

Authors:  Michiel J van Esdonk; Jacobus Burggraaf; Marion Dehez; Piet H van der Graaf; Jasper Stevens
Journal:  J Pharmacokinet Pharmacodyn       Date:  2020-04-04       Impact factor: 2.745

  4 in total

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