Literature DB >> 30327356

Prediabetes Is Associated With Structural Brain Abnormalities: The Maastricht Study.

Marnix J M van Agtmaal1,2, Alfons J H M Houben3,2, Vera de Wit3, Ronald M A Henry3,2,4, Nicolaas C Schaper3,2, Pieter C Dagnelie3,5,6, Carla J van der Kallen3,2, Annemarie Koster5,7, Simone J Sep5,8, Abraham A Kroon3,2, Jacobus F A Jansen9, Paul A Hofman9, Walter H Backes9,10, Miranda T Schram3,2,4, Coen D A Stehouwer3,2.   

Abstract

OBJECTIVE: Structural brain abnormalities are key risk factors for brain diseases, such as dementia, stroke, and depression, in type 2 diabetes. It is unknown whether structural brain abnormalities already occur in prediabetes. Therefore, we investigated whether both prediabetes and type 2 diabetes are associated with lacunar infarcts (LIs), white matter hyperintensities (WMHs), cerebral microbleeds (CMBs), and brain atrophy. RESEARCH DESIGN AND METHODS: We used data from 2,228 participants (1,373 with normal glucose metabolism [NGM], 347 with prediabetes, and 508 with type 2 diabetes (oversampled); mean age 59.2 ± 8.2 years; 48.3% women) of the Maastricht Study, a population-based cohort study. Diabetes status was determined with an oral glucose tolerance test. Brain imaging was performed with 3 Tesla MRI. Results were analyzed with multivariable logistic and linear regression analyses.
RESULTS: Prediabetes and type 2 diabetes were associated with the presence of LIs (odds ratio 1.61 [95% CI 0.98-2.63] and 1.67 [1.04-2.68], respectively; P trend = 0.027), larger WMH (β 0.07 log10-transformed mL [log-mL] [95% CI 0.00-0.15] and 0.21 log-mL [0.14-0.28], respectively; P trend <0.001), and smaller white matter volumes (β -4.0 mL [-7.3 to -0.6] and -7.2 mL [-10.4 to -4.0], respectively; P trend <0.001) compared with NGM. Prediabetes was not associated with gray matter volumes or the presence of CMBs.
CONCLUSIONS: Prediabetes is associated with structural brain abnormalities, with further deterioration in type 2 diabetes. These results indicate that, in middle-aged populations, structural brain abnormalities already occur in prediabetes, which may suggest that the treatment of early dysglycemia may contribute to the prevention of brain diseases.
© 2018 by the American Diabetes Association.

Entities:  

Mesh:

Year:  2018        PMID: 30327356     DOI: 10.2337/dc18-1132

Source DB:  PubMed          Journal:  Diabetes Care        ISSN: 0149-5992            Impact factor:   19.112


  20 in total

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Journal:  JAMA Neurol       Date:  2019-05-01       Impact factor: 18.302

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5.  Association of Plasma Neurofilament Light Chain With Glycaemic Control and Insulin Resistance in Middle-Aged Adults.

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6.  (Pre)diabetes, glycemia, and daily glucose variability are associated with retinal nerve fiber layer thickness in The Maastricht Study.

Authors:  Frank C T van der Heide; Yuri D Foreman; Iris W M Franken; Ronald M A Henry; Abraham A Kroon; Pieter C Dagnelie; Simone J P M Eussen; Tos T J M Berendschot; Jan S A G Schouten; Carroll A B Webers; Miranda T Schram; Carla J H van der Kallen; Marleen M J van Greevenbroek; Anke Wesselius; Casper G Schalkwijk; Nicolaas C Schaper; Martijn C G J Brouwers; Coen D A Stehouwer
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7.  Liraglutide improves memory in obese patients with prediabetes or early type 2 diabetes: a randomized, controlled study.

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Review 9.  Extracellular vesicles-incorporated microRNA signature as biomarker and diagnosis of prediabetes state and its complications.

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10.  Neuritin inhibits astrogliosis to ameliorate diabetic cognitive dysfunction.

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Journal:  J Mol Endocrinol       Date:  2021-04       Impact factor: 5.098

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