Literature DB >> 30303141

Prevalence estimation of microsatellite instability in colorectal cancers using tissue microarray based methods - A tertiary care center experience.

Sonali Susmita Nayak1, Paromita Roy1, Neeraj Arora2, Indu Arun1, Manas Kumar Roy3, Sudeep Banerjee3, Indranil Mallick4, Mohandas K Mallath5.   

Abstract

AIM: Microsatellite instability (MSI) pathway is known to be implicated in carcinogenesis of 15% colorectal carcinomas (CRC), including 2%-3% of cases of Lynch syndrome, as per western literature. MSI status has important prognostic and therapeutic implications. The prevalence of MSI in Indian CRC patients is unknown. We aimed to determine the prevalence by studying 231 consecutive unselected cases of CRC.
METHODS: Tissue microarrays using duplicate cores per case for 141 cases, and whole tissue sections for 90 cases, were used. Immunohistochemistry with four mismatch repair (MMR) markers - MLH1, MSH2, MSH6, and PMS2 was performed. Molecular analysis for MSI status was performed in 18 randomly selected cases. Correlation with various clinical and histopathological features was done using univariate and multivariate analysis.
RESULTS: Loss of MMR immunohistochemical (IHC) was seen in 53/231 cases, i.e. 22.94% (95% confidence interval 17.52%-28.36%). MLH1-PMS2 dual loss comprised 13.9%, MSH2-MSH6 7.4%, and isolated PMS2 loss in 1.73% of cases. Univariate analysis showed significant association with age (<60 years), right-sided tumor location, histologic type, high grade, the presence of severe intratumoral lymphocytic (ITL) and peri-tumoral lymphocytic response, and N0 nodal stage. On multivariate analysis, independent variables were age < 60 years, right-sided location, and severe ITL. Molecular testing for MSI corroborated with the IHC results.
CONCLUSION: The study results show a slightly higher prevalence of MSI-H phenotype, compared to Western literature, stressing the need for more widespread testing for better clinical management and identification of possible hereditary colon cancer syndrome.

Entities:  

Keywords:  Colorectal carcinoma; microsatellite instability; mismatch repair protein

Mesh:

Year:  2018        PMID: 30303141     DOI: 10.4103/IJPM.IJPM_430_17

Source DB:  PubMed          Journal:  Indian J Pathol Microbiol        ISSN: 0377-4929            Impact factor:   0.740


  5 in total

1.  Evaluating morphological features for predicting microsatellite instability status in colorectal cancer.

Authors:  Ajay Malik; Jasvinder Kaur Bhatia; Kavita Sahai; Dibyajyoti Boruah; A Sharma
Journal:  Med J Armed Forces India       Date:  2021-07-15

2.  Response to Immunotherapy in Combination With Mitotane in Patients With Metastatic Adrenocortical Cancer.

Authors:  Lia Head; Katja Kiseljak-Vassiliades; Toshimasa J Clark; Hilary Somerset; Jonathan King; Christopher Raeburn; Maria Albuja-Cruz; Michael Weyant; Joseph Cleveland; Margaret E Wierman; Stephen Leong
Journal:  J Endocr Soc       Date:  2019-10-11

3.  Co-inhibitor expression on tumor infiltrating and splenic lymphocytes after dual checkpoint inhibition in a microsatellite stable model of colorectal cancer.

Authors:  Ryan J Slovak; Hong-Jai Park; William M Kamp; Johannes M Ludwig; Insoo Kang; Hyun S Kim
Journal:  Sci Rep       Date:  2021-03-26       Impact factor: 4.379

4.  Mismatch repair deficiency occurs very rarely in seminomas.

Authors:  David Dum; Stefan Steurer; Ronald Simon; Pia Victoria Zimmermann; Eike Burandt; Till Sebastian Clauditz; Margit Fisch; Michael Rink; Roland Dahlem; Wolfgang Höppner; Henrik Zecha; Ousman Doh; Cord Matthies; Waldemar Wilczak; Guido Sauter; Christoph Fraune
Journal:  Transl Androl Urol       Date:  2021-03

5.  Identification of colorectal cancers with defective DNA damage repair by immunohistochemical profiling of mismatch repair proteins, CDX2 and BRCA1.

Authors:  Savitha Rajarajan; Anupama C E; Betsy Jose; Marjorie Correa; Sagar Sengupta; Jyothi S Prabhu
Journal:  Mol Clin Oncol       Date:  2020-09-01
  5 in total

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