Literature DB >> 30301590

Genetic analysis of undiagnosed ataxia-telangiectasia-like disorders.

Ayako Kashimada1, Setsuko Hasegawa1, Toshihiro Nomura1, Hiroshi Shiraku1, Kengo Moriyama1, Tomonori Suzuki1, Keisuke Nakajima1, Tomoko Mizuno1, Kohsuke Imai1, Yuji Sugawara1, Tomohiro Morio1, Satoko Kumada2, Masatoshi Takagi3.   

Abstract

OBJECTIVES: Defects in DNA damage responses or repair mechanisms cause numerous rare inherited diseases, referred to as "DNA-repair defects" or "DNA damage deficiency", characterized by neurodegeneration, immunodeficiency, and/or cancer predisposition. Early accurate diagnosis is important for informing appropriate clinical management; however, diagnosis is frequently challenging and can be delayed, due to phenotypic heterogeneity. Comprehensive genomic analysis could overcome this disadvantage. The objectives of this study were to determine the prevalence of ataxia-telangiectasia (A-T) and A-T-like DNA-repair defects in Japan and to determine the utility of comprehensive genetic testing of presumptively diagnosed patients in facilitating early diagnosis.
METHODS: A nationwide survey of diseases presumably caused by DNA-repair defects, including A-T, was performed. Additionally, comprehensive next-generation sequencing (NGS) analysis, targeting known disease-causing genes, was conducted.
RESULTS: Sixty-three patients with A-T or other diseases with characteristics of DNA-repair defects were identified. Thirty-four patients were genetically or clinically definitively diagnosed with A-T (n = 22) or other DNA-repair defects (n = 12). Genetic analysis of 17 presumptively diagnosed patients revealed one case of ataxia with oculomotor apraxia type 1 (AOA1); one ataxia with oculomotor apraxia type 2 (AOA2); two types of autosomal dominant spinocerebellar ataxia (SCA5, SCA29); two CACNA1A-related ataxias; one microcephaly with or without chorioretinopathy, lymphedema, or mental retardation (MCLMR); and one autosomal dominant KIF1A-related disorder with intellectual deficit, cerebellar atrophy, spastic paraparesis, and optic nerve atrophy. The diagnostic yield was 58.8%.
CONCLUSION: Comprehensive genetic analysis of targeted known disease-causing genes by NGS is a powerful diagnostic tool for subjects with indistinguishable neurological phenotypes resembling DNA-repair defects.
Copyright © 2018 The Japanese Society of Child Neurology. Published by Elsevier B.V. All rights reserved.

Entities:  

Keywords:  Ataxia-telangiectasia; Cerebellar ataxia; DNA-repair defects; Microcephaly; Next-generation sequencing; Spinocerebellar ataxia

Mesh:

Year:  2018        PMID: 30301590     DOI: 10.1016/j.braindev.2018.09.007

Source DB:  PubMed          Journal:  Brain Dev        ISSN: 0387-7604            Impact factor:   1.961


  8 in total

1.  Channelopathies Are a Frequent Cause of Genetic Ataxias Associated with Cerebellar Atrophy.

Authors:  Laurence Gauquelin; Taila Hartley; Mark Tarnopolsky; David A Dyment; Bernard Brais; Michael T Geraghty; Martine Tétreault; Sohnee Ahmed; Samantha Rojas; Karine Choquet; Jacek Majewski; François Bernier; Allan Micheil Innes; Guy Rouleau; Oksana Suchowersky; Kym M Boycott; Grace Yoon
Journal:  Mov Disord Clin Pract       Date:  2020-09-29

2.  Monoallelic KIF1A-related disorders: a multicenter cross sectional study and systematic literature review.

Authors:  Stefania Della Vecchia; Alessandra Tessa; Claudia Dosi; Jacopo Baldacci; Rosa Pasquariello; Antonella Antenora; Guja Astrea; Maria Teresa Bassi; Roberta Battini; Carlo Casali; Ettore Cioffi; Greta Conti; Giovanna De Michele; Anna Rita Ferrari; Alessandro Filla; Chiara Fiorillo; Carlo Fusco; Salvatore Gallone; Chiara Germiniasi; Renzo Guerrini; Shalom Haggiag; Diego Lopergolo; Andrea Martinuzzi; Federico Melani; Andrea Mignarri; Elena Panzeri; Antonella Pini; Anna Maria Pinto; Francesca Pochiero; Guido Primiano; Elena Procopio; Alessandra Renieri; Romina Romaniello; Cristina Sancricca; Serenella Servidei; Carlotta Spagnoli; Chiara Ticci; Anna Rubegni; Filippo Maria Santorelli
Journal:  J Neurol       Date:  2021-09-06       Impact factor: 4.849

Review 3.  Polymerases and DNA Repair in Neurons: Implications in Neuronal Survival and Neurodegenerative Diseases.

Authors:  Xiaoling Li; Guanghui Cao; Xiaokang Liu; Tie-Shan Tang; Caixia Guo; Hongmei Liu
Journal:  Front Cell Neurosci       Date:  2022-06-30       Impact factor: 6.147

Review 4.  Beyond Typical Ataxia Telangiectasia: How to Identify the Ataxia Telangiectasia-Like Disorders.

Authors:  Ivana Rocha Raslan; Paula Camila Alves de Assis Pereira Matos; Vinícius Boaratti Ciarlariello; Karyme Hussein Daghastanli; Augusto Bragança Reis Rosa; Juliana Harumi Arita; Carolina Sanchez Aranda; Orlando Graziani Povoas Barsottini; José Luiz Pedroso
Journal:  Mov Disord Clin Pract       Date:  2020-11-19

Review 5.  Markers of Toxicity and Response to Radiation Therapy in Patients With Prostate Cancer.

Authors:  Nicola J Nasser; Jonathan Klein; Abed Agbarya
Journal:  Adv Radiat Oncol       Date:  2020-10-27

6.  Genotype and defects in microtubule-based motility correlate with clinical severity in KIF1A-associated neurological disorder.

Authors:  Lia Boyle; Lu Rao; Simranpreet Kaur; Xiao Fan; Caroline Mebane; Laura Hamm; Andrew Thornton; Jared T Ahrendsen; Matthew P Anderson; John Christodoulou; Arne Gennerich; Yufeng Shen; Wendy K Chung
Journal:  HGG Adv       Date:  2021-01-30

Review 7.  Calcium channelopathies and intellectual disability: a systematic review.

Authors:  Miriam Kessi; Baiyu Chen; Jing Peng; Fangling Yan; Lifen Yang; Fei Yin
Journal:  Orphanet J Rare Dis       Date:  2021-05-13       Impact factor: 4.123

8.  A Novel SETX Mutation in a Taiwanese Patient with Autosomal Recessive Cerebellar Ataxia Detected by Targeted Next-Generation Sequencing, and a Literature Review.

Authors:  Ping-I Chiang; Ting-Wei Liao; Chiung-Mei Chen
Journal:  Brain Sci       Date:  2022-01-28
  8 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.