Literature DB >> 30275189

miR-3148 Is a Novel Onco-microRNA that Potentiates Tumor Growth In Vivo.

Takaki Akamine1,2, Yosuke Morodomi1,2, Yui Harada2, Koji Teraishi1,2, Tetsuzo Tagawa1, Tatsuro Okamoto1, Yoshihiko Maehara1, Yoshikazu Yonemitsu3.   

Abstract

BACKGROUND/AIM: Alterations of microRNA expression in three-dimensional spheroids were examined to identify novel microRNAs that might be associated with tumorigenesis.
MATERIALS AND METHODS: Using microRNA microarray analysis, we screened for microRNAs that were dramatically up-regulated inside three-dimensional spheroids in genetically-modified HCT116 human colon cancer cells expressing Copepoda Green Fluorescent Protein under hypoxia.
RESULTS: miR-3148 was identified as a possible candidate onco-microRNA. A growth advantage of HCT116 cells stably expressing miR-3148 (HCT116-miR3148) was observed compared to parental cells in vivo, but not in vitro. Additionally, no change in growth under hypoxic or starvation conditions was seen in these cells cultured two-dimensionally; however, HCT116-miR3148 cells maintained as three-dimensional spheroids were highly resistant to hypoxic conditions. HCT116-miR3148 cells were more sensitive to mitogen-activated protein kinase (MAPK) kinase inhibitors and extracellular signal-regulated kinase (ERK) inhibitors.
CONCLUSION: MiR-3148 may be a novel onco-microRNA that protects cancer cells from serious stress conditions through the MAPK/ERK pathway, especially in vivo. Copyright
© 2018, International Institute of Anticancer Research (Dr. George J. Delinasios), All rights reserved.

Entities:  

Keywords:  MicroRNA-3148; colon cancer; onco-miR; spheroid

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Year:  2018        PMID: 30275189     DOI: 10.21873/anticanres.12906

Source DB:  PubMed          Journal:  Anticancer Res        ISSN: 0250-7005            Impact factor:   2.480


  2 in total

1.  MicroRNA-3148 acts as molecular switch promoting malignant transformation and adipocytic differentiation of immortalized human bone marrow stromal cells via direct targeting of the SMAD2/TGFβ pathway.

Authors:  Radhakrishnan Vishnubalaji; Ramesh Elango; Muthurangan Manikandan; Abdul-Aziz Siyal; Dalia Ali; Ammar Al-Rikabi; Dana Hamam; Rimi Hamam; Hicham Benabdelkamel; Afshan Masood; Ibrahim O Alanazi; Assim A Alfadda; Musaad Alfayez; Abdullah Aldahmash; Moustapha Kassem; Nehad M Alajez
Journal:  Cell Death Discov       Date:  2020-09-01

2.  OTOP2, Inversely Modulated by miR-3148, Inhibits CRC Cell Migration, Proliferation and Epithelial-Mesenchymal Transition: Evidence from Bioinformatics Data Mining and Experimental Verification.

Authors:  Shuai Guo; Yang Sun
Journal:  Cancer Manag Res       Date:  2022-04-07       Impact factor: 3.989

  2 in total

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