Literature DB >> 30244530

Small 4p16.3 deletions: Three additional patients and review of the literature.

Laura Bernardini1, Francesca C Radio2, Fabio Acquaviva3, Cristina Gorgone4, Diana Postorivo5, Barbara Torres1, Viola Alesi2, Monia Magliozzi2, Fortunato Lonardo3, Matteo Della Monica6, Anna M Nardone5, Claudia Cesario2, Teresa Mattina4, Gioacchino Scarano3, Bruno Dallapiccola2, Maria C Digilio2, Antonio Novelli2.   

Abstract

Wolf-Hirschhorn syndrome is a well-defined disorder due to 4p16.3 deletion, characterized by distinct facial features, intellectual disability, prenatal and postnatal growth retardation, and seizures. Genotype-phenotype correlations based on differently sized deletions have been attempted, and some candidate genes have been suggested. We report on clinical characteristics of three patients with pure interstitial submicroscopic 4p16.3 deletions, ranging in size from 68 to 166 kb, involving WHSCR1 and/or part of WHSCR2, and review published cases with overlapping 4p16.3 losses. The present study highlights a major role of NSD2 gene in the pathogenesis of the WHS main features and predicts that loss-of-function mutations affecting NSD2 gene could result in microcephaly, prenatal and postnatal growth retardation, psychomotor and language delay, and craniofacial features. Absent seizures in all subjects corroborate the suggestion that this specific feature is causally linked with at least one additional causative gene. Finally, we suggest that mir-943 could play a role in the pathogenesis of CHD in some of these patients.
© 2018 Wiley Periodicals, Inc.

Entities:  

Keywords:  CHD; NSD2 gene; Wolf-Hirschhorn syndrome; mir-943

Mesh:

Year:  2018        PMID: 30244530     DOI: 10.1002/ajmg.a.40512

Source DB:  PubMed          Journal:  Am J Med Genet A        ISSN: 1552-4825            Impact factor:   2.802


  4 in total

1.  De Novo Inverted Duplication Deletion of 4p in a 14-Week-Old Male Fetus Aborted Due to Multiple Anomalies.

Authors:  Paolo Fontana; Laura Bernardini; Cinzia Lombardi; Maria Grazia Giuffrida; Maria Ciavarella; Anna Capalbo; Marianna Maioli; Francesca Scarano; Giuseppina Cantalupo; Mariateresa Falco; Gioacchino Scarano; Fortunato Lonardo
Journal:  J Pediatr Genet       Date:  2020-06-19

2.  Wolf-Hirschhorn Syndrome-Associated Genes Are Enriched in Motile Neural Crest Cells and Affect Craniofacial Development in Xenopus laevis.

Authors:  Alexandra Mills; Elizabeth Bearce; Rachael Cella; Seung Woo Kim; Megan Selig; Sangmook Lee; Laura Anne Lowery
Journal:  Front Physiol       Date:  2019-04-12       Impact factor: 4.566

Review 3.  From Wolf-Hirschhorn syndrome to NSD2 haploinsufficiency: a shifting paradigm through the description of a new case and a review of the literature.

Authors:  Luisa Cortellazzo Wiel; Irene Bruno; Egidio Barbi; Fabio Sirchia
Journal:  Ital J Pediatr       Date:  2022-05-12       Impact factor: 3.288

4.  Loss-of-function and missense variants in NSD2 cause decreased methylation activity and are associated with a distinct developmental phenotype.

Authors:  Paolo Zanoni; Katharina Steindl; Deepanwita Sengupta; Or Gozani; Anita Rauch; Pascal Joset; Angela Bahr; Heinrich Sticht; Mariarosaria Lang-Muritano; Conny M A van Ravenswaaij-Arts; Marwan Shinawi; Marisa Andrews; Tania Attie-Bitach; Isabelle Maystadt; Newell Belnap; Valerie Benoit; Geoffroy Delplancq; Bert B A de Vries; Sarah Grotto; Didier Lacombe; Austin Larson; Jeroen Mourmans; Katrin Õunap; Giulia Petrilli; Rolph Pfundt; Keri Ramsey; Lot Snijders Blok; Vassilis Tsatsaris; Antonio Vitobello; Laurence Faivre; Patricia G Wheeler; Marijke R Wevers; Monica Wojcik; Markus Zweier
Journal:  Genet Med       Date:  2021-05-03       Impact factor: 8.822

  4 in total

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