Literature DB >> 30096316

Classified status of smoking and quitting has different associations with dyslipidemia in residents in northeast China.

Jikang Shi1, Ye Bai1, Shuang Qiu1, Yong Li1, Changgui Kou1, Yuchun Tao1, Qing Zhen1, Yulu Gu1, Yaqin Yu1, Kaixin Zhang1, Yi Cheng2, Yawen Liu3.   

Abstract

BACKGROUND: Various smoking status and high prevalence of dyslipidemia in residents exist in northeast China. However, associations of dyslipidemia with smoking status remain unclear.
METHODS: A total of 17,114 participants selected by a multistage stratified cluster random sampling design were enrolled from a cross-sectional study conducted in northeast China. Associations of dyslipidemia with smoking/quitting status (smoking amount, smoking duration, and quitting duration) were investigated using multiple logistic regression.
RESULTS: Prevalence (39.2%) of dyslipidemia existed in residents in northeast China. Smoking amount was associated with dyslipidemia (1-10 cigarettes daily: OR = 1.19, 95% CI: 1.08-1.32; 11-20 cigarettes daily: OR = 1.29, 95% CI: 1.16-1.42; and over 20 cigarettes daily: OR = 1.51, 95% CI: 1.25-1.83). Smoking duration was associated with dyslipidemia risk (6-10 years: OR = 1.75, 95% CI: 1.51-2.03; 11-15 years: OR = 1.85, 95% CI: 1.51-2.26; and ≥15 years: OR = 1.12, 95% CI: 1.02-1.23). Quitting duration (1-5 years) was associated with dyslipidemia (OR = 1.26, 95% CI: 1.07-1.48); however, we found no statistically significant associations between dyslipidemia and quitting duration (over 6 years).
CONCLUSIONS: Dyslipidemia risk is positively associated with smoking/quitting status. Smoking amount and smoking duration may co-determine dyslipidemia risk, and quitting duration (>6 years) is necessary for reducing dyslipidemia risk.
Copyright © 2018 Elsevier B.V. All rights reserved.

Entities:  

Keywords:  Dyslipidemia; Quitting duration; Smoking amount; Smoking duration

Mesh:

Year:  2018        PMID: 30096316     DOI: 10.1016/j.cca.2018.08.001

Source DB:  PubMed          Journal:  Clin Chim Acta        ISSN: 0009-8981            Impact factor:   3.786


  2 in total

1.  Cigarette smoke exposure impairs lipid metabolism by decreasing low-density lipoprotein receptor expression in hepatocytes.

Authors:  Baitao Ma; Yunfei Chen; Xuebin Wang; Rui Zhang; Shuai Niu; Leng Ni; Xiao Di; Qin Han; Changwei Liu
Journal:  Lipids Health Dis       Date:  2020-05-08       Impact factor: 3.876

2.  Cigarette smoke extract stimulates PCSK9 production in HepG2 cells via ROS/NF‑κB signaling.

Authors:  Baitao Ma; Xuebin Wang; Rui Zhang; Shuai Niu; Zhihua Rong; Leng Ni; Xiao Di; Qin Han; Changwei Liu
Journal:  Mol Med Rep       Date:  2021-03-24       Impact factor: 2.952

  2 in total

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