Literature DB >> 30093155

Application of a neuroscience research model to study neuroprotection in children with congenital heart disease.

Nobuyuki Ishibashi1, Richard A Jonas2.   

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Year:  2018        PMID： 30093155      PMCID： PMC6812669          DOI： 10.1016/j.jtcvs.2018.06.067

Source DB:  PubMed          Journal:  J Thorac Cardiovasc Surg        ISSN： 0022-5223            Impact factor:   5.209


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  9 in total

1.  Minocycline alleviates hypoxic-ischemic injury to developing oligodendrocytes in the neonatal rat brain.

Authors:  Z Cai; S Lin; L-W Fan; Y Pang; P G Rhodes
Journal:  Neuroscience       Date:  2005-11-14       Impact factor: 3.590

2.  De novo mutations in congenital heart disease with neurodevelopmental and other congenital anomalies.

Authors:  Jason Homsy; Samir Zaidi; Yufeng Shen; James S Ware; Kaitlin E Samocha; Konrad J Karczewski; Steven R DePalma; David McKean; Hiroko Wakimoto; Josh Gorham; Sheng Chih Jin; John Deanfield; Alessandro Giardini; George A Porter; Richard Kim; Kaya Bilguvar; Francesc López-Giráldez; Irina Tikhonova; Shrikant Mane; Angela Romano-Adesman; Hongjian Qi; Badri Vardarajan; Lijiang Ma; Mark Daly; Amy E Roberts; Mark W Russell; Seema Mital; Jane W Newburger; J William Gaynor; Roger E Breitbart; Ivan Iossifov; Michael Ronemus; Stephan J Sanders; Jonathan R Kaltman; Jonathan G Seidman; Martina Brueckner; Bruce D Gelb; Elizabeth Goldmuntz; Richard P Lifton; Christine E Seidman; Wendy K Chung
Journal:  Science       Date:  2015-12-04       Impact factor: 47.728

3.  Inhibition of microglial activation by minocycline reduced preoligodendrocyte injury in a neonatal rat brain slice model.

Authors:  Junrong Huang; Gang Liu; Bowen Shi; Guochen Shi; Xiaomin He; Zhaohui Lu; Jinghao Zheng; Haibo Zhang; Huiwen Chen; Zhongqun Zhu
Journal:  J Thorac Cardiovasc Surg       Date:  2018-07-18       Impact factor: 5.209

Review 4.  Neurodevelopmental Abnormalities and Congenital Heart Disease: Insights Into Altered Brain Maturation.

Authors:  Paul D Morton; Nobuyuki Ishibashi; Richard A Jonas
Journal:  Circ Res       Date:  2017-03-17       Impact factor: 17.367

5.  Abnormal brain development in newborns with congenital heart disease.

Authors:  Steven P Miller; Patrick S McQuillen; Shannon Hamrick; Duan Xu; David V Glidden; Natalie Charlton; Tom Karl; Anthony Azakie; Donna M Ferriero; A James Barkovich; Daniel B Vigneron
Journal:  N Engl J Med       Date:  2007-11-08       Impact factor: 91.245

6.  New white matter brain injury after infant heart surgery is associated with diagnostic group and the use of circulatory arrest.

Authors:  John Beca; Julia K Gunn; Lee Coleman; Ayton Hope; Peter W Reed; Rodney W Hunt; Kirsten Finucane; Christian Brizard; Brieana Dance; Lara S Shekerdemian
Journal:  Circulation       Date:  2013-01-31       Impact factor: 29.690

7.  Prolonged White Matter Inflammation After Cardiopulmonary Bypass and Circulatory Arrest in a Juvenile Porcine Model.

Authors:  Ludmila Korotcova; Sonali Kumar; Kota Agematsu; Paul D Morton; Richard A Jonas; Nobuyuki Ishibashi
Journal:  Ann Thorac Surg       Date:  2015-07-27       Impact factor: 4.330

8.  Hypoxia diminishes the protective function of white-matter astrocytes in the developing brain.

Authors:  Kota Agematsu; Ludmila Korotcova; Paul D Morton; Vittorio Gallo; Richard A Jonas; Nobuyuki Ishibashi
Journal:  J Thorac Cardiovasc Surg       Date:  2015-09-26       Impact factor: 5.209

9.  Effects of preoperative hypoxia on white matter injury associated with cardiopulmonary bypass in a rodent hypoxic and brain slice model.

Authors:  Kota Agematsu; Ludmila Korotcova; Joseph Scafidi; Vittorio Gallo; Richard A Jonas; Nobuyuki Ishibashi
Journal:  Pediatr Res       Date:  2014-01-31       Impact factor: 3.756
  9 in total
  1 in total

1.  Part II. Comparison of Neurodevelopmental Outcomes Between Normothermic and Hypothermic Pediatric Cardiopulmonary Bypass.

Authors:  Claire E Hannon; Zachary Osman; Cathy Grant; Emma M L Chung; Antonio F Corno
Journal:  Front Pediatr       Date:  2019-11-05       Impact factor: 3.418
  1 in total

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