H A García-Perdomo1,2,3, J Zapata-Copete2,3, C A Rojas-Cerón1. 1. School of Medicine - Universidad del Valle, Cali, Colombia. 2. Epidemiology Department, Universidad Libre, Cali, Colombia. 3. UROGIV Research Group Universidad del Valle, Cali, Colombia.
Abstract
AIMS: To determine the association between the sleep duration and the risk of all-cause mortality in adults. METHODS: A search strategy was conducted in the MEDLINE, CENTRAL, EMBASE and LILACS databases. Searches were also conducted in other databases and unpublished literature. Cohort studies were included without language, time or setting restrictions. The risk of bias was evaluated with a modified Cochrane Collaboration's tool. An analysis of random effects was conducted. The primary outcome was all-cause mortality. The measure of the effect was the risk difference (RD) with a 95% confidence interval (CI). The planned comparisons were 7-9 h of sleep v. <7 h and the same reference v. >9 h. RESULTS: Thirty-nine studies were included in our qualitative analysis, regarding the quantitative analysis, 19 studies were included in <7 v. 7-9 h analysis, and 18 studies in the >9 v. 7-9 h. A low risk of bias was shown for most of the study items. The overall RD for all-cause mortality was 0.09 (95% CI 0.07-0.11) favouring the >9 h group compared with our reference. In contrast, no differences were found between the <7 h and the reference sleep duration groups (RD 0.00, 95% CI 0.00-0.01). CONCLUSION: We found a probable association of long sleep duration and higher mortality; however, it could reflect an underlying systemic or neurological disease that cause sleep fragmentation, deterioration in quality and micro-awakenings.
AIMS: To determine the association between the sleep duration and the risk of all-cause mortality in adults. METHODS: A search strategy was conducted in the MEDLINE, CENTRAL, EMBASE and LILACS databases. Searches were also conducted in other databases and unpublished literature. Cohort studies were included without language, time or setting restrictions. The risk of bias was evaluated with a modified Cochrane Collaboration's tool. An analysis of random effects was conducted. The primary outcome was all-cause mortality. The measure of the effect was the risk difference (RD) with a 95% confidence interval (CI). The planned comparisons were 7-9 h of sleep v. <7 h and the same reference v. >9 h. RESULTS: Thirty-nine studies were included in our qualitative analysis, regarding the quantitative analysis, 19 studies were included in <7 v. 7-9 h analysis, and 18 studies in the >9 v. 7-9 h. A low risk of bias was shown for most of the study items. The overall RD for all-cause mortality was 0.09 (95% CI 0.07-0.11) favouring the >9 h group compared with our reference. In contrast, no differences were found between the <7 h and the reference sleep duration groups (RD 0.00, 95% CI 0.00-0.01). CONCLUSION: We found a probable association of long sleep duration and higher mortality; however, it could reflect an underlying systemic or neurological disease that cause sleep fragmentation, deterioration in quality and micro-awakenings.
Authors: M Kojima; K Wakai; T Kawamura; A Tamakoshi; R Aoki; Y Lin; T Nakayama; H Horibe; N Aoki; Y Ohno Journal: J Epidemiol Date: 2000-03 Impact factor: 3.211
Authors: David Martínez-Gómez; Pilar Guallar-Castillón; Luz M León-Muñoz; Esther López-García; Fernando Rodríguez-Artalejo Journal: BMC Med Date: 2013-02-22 Impact factor: 8.775
Authors: Yue Leng; Francesco P Cappuccio; Nick W J Wainwright; Paul G Surtees; Robert Luben; Carol Brayne; Kay-Tee Khaw Journal: Neurology Date: 2015-02-25 Impact factor: 9.910
Authors: Aline Silva-Costa; Lucia Rotenberg; Aline A Nobre; Dora Chor; Estela M Aquino; Enirtes C Melo; Sandhi M Barreto; Maria Inês Schmidt; Rosane H Griep Journal: Arch Public Health Date: 2020-05-29