Literature DB >> 30015394

The serum KL-6 levels in untreated idiopathic pulmonary fibrosis can naturally decline in association with disease progression.

Hiroshi Ishii1, Hisako Kushima1, Yoshiaki Kinoshita1, Masaki Fujita1, Kentaro Watanabe1.   

Abstract

BACKGROUND: Serum KL-6 is known to be a useful biomarker for the diagnosis and assessment of the disease activity in idiopathic pulmonary fibrosis (IPF). However, some patients with untreated IPF present with a serial decline in their serum KL-6 levels, despite showing clinical progression.
OBJECTIVES: The aim of the present study was to evaluate the association between the time course of the serum KL-6 levels and the clinical course of patients with untreated IPF.
METHODS: We conducted a retrospective study of 59 patients who met the diagnostic criteria for IPF, who regularly underwent laboratory tests, including the measurement of KL-6 and pulmonary function tests, and who were untreated during the follow-up period.
RESULTS: The rates of decline in comparison to the baseline FVC values were comparable between survivors (n = 30) and non-survivors (n = 29); however, the rates of change in comparison to the baseline KL-6 levels were heterogeneous in all subjects, and the KL-6 levels of the non-survivors were observed to decline significantly more frequently in comparison to the survivors (P = .001). Patients with a higher breathlessness scale, lower pulmonary functions and higher levels of baseline KL-6 were predisposed to demonstrate a decline in the serum KL-6 levels during the untreated period.
CONCLUSIONS: A reduction in the serum KL-6 levels of patients with untreated IPF may not mean that the disease activity has subsided. Serum KL-6 levels can naturally decline in association with disease progression.
© 2018 John Wiley & Sons Ltd.

Entities:  

Keywords:  KL-6; forced vital capacity; idiopathic pulmonary fibrosis; survival

Mesh:

Substances:

Year:  2018        PMID: 30015394     DOI: 10.1111/crj.12946

Source DB:  PubMed          Journal:  Clin Respir J        ISSN: 1752-6981            Impact factor:   2.570


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