Literature DB >> 29989318

TIMP-1 association with collagen type I overproduction in hereditary gingival fibromatosis.

Katarzyna Gawron1, Anna Ochała-Kłos2, Zuzanna Nowakowska1, Grzegorz Bereta1, Katarzyna Łazarz-Bartyzel3, Aleksander M Grabiec1, Paweł Plakwicz4, Renata Górska4, Andrzej Fertala5, Maria Chomyszyn-Gajewska3, Jan Potempa1,6.   

Abstract

OBJECTIVES: To investigate the processes associated with the excessive production of collagen I in hereditary gingival fibromatosis (HGF).
MATERIALS AND METHODS: Three HGF subjects and five controls were enrolled in the study. Histomorphological and immunohistological analyses were performed on gingival tissues. The expression of heat-shock protein 47 (HSP47), collagen I, transforming growth factor-β1 (TGF-β1), connective tissue growth factor (CTGF), matrix metalloproteinase-1 (MMP-1) and tissue inhibitor of matrix metalloproteinase-1 (TIMP-1) by gingival fibroblasts isolated from HGF and controls was analysed using qRT-PCR, Western blotting and ELISA.
RESULTS: Considerable accumulation of fibrotic fibrils and increased synthesis of HSP47 were noted in HGF gingival tissues. The synthesis of collagen I, HSP47, TGF-β1, CTGF and TIMP-1 was significantly elevated in HGF gingival fibroblasts compared with controls, while the production of MMP-1 was decreased.
CONCLUSIONS: We report that fibrosis in HGF gingival tissues is associated with increased synthesis of HSP47. This finding was confirmed by an in vitro study, where excessive production of collagen I was associated with increased synthesis of HSP47, TGF-β1 and CTGF by HGF gingival fibroblasts. Moreover, the shift in the TIMP-1/MMP-1 ratio identifies increased synthesis of TIMP-1 as one of the processes associated with collagen I overproduction in HGF fibroblasts.
© 2018 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd. All rights reserved.

Entities:  

Keywords:  TIMP-1; collagen I; fibrosis; hereditary gingival fibromatosis

Mesh:

Substances:

Year:  2018        PMID: 29989318     DOI: 10.1111/odi.12938

Source DB:  PubMed          Journal:  Oral Dis        ISSN: 1354-523X            Impact factor:   3.511


  4 in total

1.  BET Bromodomain Inhibitors Suppress Inflammatory Activation of Gingival Fibroblasts and Epithelial Cells From Periodontitis Patients.

Authors:  Anna Maksylewicz; Agnieszka Bysiek; Katarzyna B Lagosz; Justyna M Macina; Malgorzata Kantorowicz; Grzegorz Bereta; Maja Sochalska; Katarzyna Gawron; Maria Chomyszyn-Gajewska; Jan Potempa; Aleksander M Grabiec
Journal:  Front Immunol       Date:  2019-04-30       Impact factor: 7.561

2.  The aryl hydrocarbon receptor pathway controls matrix metalloproteinase-1 and collagen levels in human orbital fibroblasts.

Authors:  Elisa Roztocil; Christine L Hammond; Mithra O Gonzalez; Steven E Feldon; Collynn F Woeller
Journal:  Sci Rep       Date:  2020-05-21       Impact factor: 4.379

3.  A novel gene ZNF862 causes hereditary gingival fibromatosis.

Authors:  Juan Wu; Dongna Chen; Hui Huang; Ning Luo; Huishuang Chen; Junjie Zhao; Yanyan Wang; Tian Zhao; Siyuan Huang; Yang Ren; Teng Zhai; Weibin Sun; Houxuan Li; Wei Li
Journal:  Elife       Date:  2022-02-10       Impact factor: 8.140

Review 4.  Clinics and genetic background of hereditary gingival fibromatosis.

Authors:  Karolina Strzelec; Agata Dziedzic; Katarzyna Łazarz-Bartyzel; Aleksander M Grabiec; Ewa Gutmajster; Tomasz Kaczmarzyk; Paweł Plakwicz; Katarzyna Gawron
Journal:  Orphanet J Rare Dis       Date:  2021-11-24       Impact factor: 4.123

  4 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.