Literature DB >> 29983411

Rituximab modulates T- and B-lymphocyte subsets and urinary CD80 excretion in patients with steroid-dependent nephrotic syndrome.

Divya Bhatia1, Aditi Sinha1, Pankaj Hari1, Shailaja Sopory2, Savita Saini1, Mamta Puraswani1, Himanshi Saini1, Dipendra K Mitra3, Arvind Bagga4.   

Abstract

BACKGROUND: Rituximab, a monoclonal antibody targeting B lymphocytes, effectively sustains remission in steroid-dependent nephrotic syndrome (SDNS). We studied its effects on lymphocyte subsets and urinary CD80 excretion (uCD80) in patients with SDNS.
METHODS: Blood and urine samples were collected from 18 SDNS patients before rituximab, and after 1 month and 1 year or at first relapse. T and B lymphocytes and uCD80 were determined by flow cytometry and ELISA, respectively.
RESULTS: Treatment was associated with reduction in counts of Th17, Th2, and memory T cells, and increased T-regulatory (Treg) cells. The Th17/Treg ratio declined from baseline (median 0.6) to 1 month (0.2, P = 0.006) and increased during relapse (0.3, P = 0.016). Ratios of Th1/Th2 cells at baseline, 1 month after rituximab, and during relapse were 7.7, 14.0 (P = 0.0102), and 8.7, respectively. uCD80 decreased 1 month following rituximab (45.5 vs. 23.0 ng/g creatinine; P = 0.0039). B lymphocytes recovered earlier in relapsers (60.0 vs.183.0 days; P < 0.001). Memory B cells were higher during relapse than remission (29.7 vs.18.0 cells/µL; P = 0.029).
CONCLUSION: Rituximab-induced sustained remission and B-cell depletion was associated with reduced numbers of Th17 and Th2 lymphocytes, and increased Treg cells; these changes reversed during relapses. Recovery of B cells and memory B cells predicted the occurrence of a relapse.

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Year:  2018        PMID: 29983411     DOI: 10.1038/s41390-018-0088-7

Source DB:  PubMed          Journal:  Pediatr Res        ISSN: 0031-3998            Impact factor:   3.756


  19 in total

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Authors:  Manuela Colucci; Rita Carsetti; Simona Cascioli; Jessica Serafinelli; Francesco Emma; Marina Vivarelli
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2.  Significance of regulatory T cells in children with idiopathic nephrotic syndrome.

Authors:  Shoji Tsuji; Tadashi Yamaguchi; Yuko Akagawa; Shohei Akagawa; Sohsaku Yamanouchi; Takahisa Kimata; Kazunari Kaneko
Journal:  J Nephrol       Date:  2022-01-28       Impact factor: 3.902

3.  Circulating plasmablasts in children with steroid-sensitive nephrotic syndrome.

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Journal:  Pediatr Nephrol       Date:  2021-10-18       Impact factor: 3.714

4.  Human or Chimeric Monoclonal Anti-CD20 Antibodies for Children with Nephrotic Syndrome: A Superiority Randomized Trial.

Authors:  Pietro Ravani; Manuela Colucci; Maurizio Bruschi; Marina Vivarelli; Michela Cioni; Armando DiDonato; Paolo Cravedi; Francesca Lugani; Francesca Antonini; Marco Prunotto; Francesco Emma; Andrea Angeletti; Gian Marco Ghiggeri
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Review 5.  Podocytopathies.

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6.  Belimumab for the treatment of children with frequently relapsing nephrotic syndrome: the BELNEPH study.

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Journal:  Pediatr Nephrol       Date:  2021-08-05       Impact factor: 3.714

Review 7.  Update on the treatment of steroid-sensitive nephrotic syndrome.

Authors:  Federica Zotta; Marina Vivarelli; Francesco Emma
Journal:  Pediatr Nephrol       Date:  2021-03-05       Impact factor: 3.714

8.  Short-term Immunopathological Changes Associated with Pulse Steroids/IVIG/Rituximab Therapy in Late Kidney Allograft Antibody Mediated Rejection.

Authors:  Kenna R Degner; Nancy A Wilson; Shannon R Reese; Sandesh Parajuli; Fahad Aziz; Neetika Garg; Maha Mohamed; Tripti Singh; Didier A Mandelbrot; Sarah E Panzer; Robert R Redfield; Kristin Van Hyfte; Weixiong Zhong; Luis G Hidalgo; Arjang Djamali
Journal:  Kidney360       Date:  2020-05-28

9.  Mitophagy-dependent macrophage reprogramming protects against kidney fibrosis.

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Journal:  JCI Insight       Date:  2019-12-05

Review 10.  Biomarkers in pediatric glomerulonephritis and nephrotic syndrome.

Authors:  Gabriel Cara-Fuentes; William E Smoyer
Journal:  Pediatr Nephrol       Date:  2021-01-03       Impact factor: 3.714

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