Literature DB >> 29955584

Can pancreatitis be treated by inhibiting Ca2+ signaling?

John A Williams1, David I Yule2.   

Abstract

Entities:  

Year:  2018        PMID： 29955584      PMCID： PMC6015945          DOI： 10.21037/atm.2017.06.07

Source DB:  PubMed          Journal:  Ann Transl Med        ISSN： 2305-5839


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  26 in total

1.  IP3 receptor types 2 and 3 mediate exocrine secretion underlying energy metabolism.

Authors:  Akira Futatsugi; Takeshi Nakamura; Maki K Yamada; Etsuko Ebisui; Kyoko Nakamura; Keiko Uchida; Tetsuya Kitaguchi; Hiromi Takahashi-Iwanaga; Tetsuo Noda; Jun Aruga; Katsuhiko Mikoshiba
Journal:  Science       Date:  2005-09-30       Impact factor: 47.728

2.  A prospective cohort study on the association between coffee drinking and risk of non-gallstone-related acute pancreatitis.

Authors:  Viktor Oskarsson; Omid Sadr-Azodi; Nicola Orsini; Alicja Wolk
Journal:  Br J Nutr       Date:  2016-03-18       Impact factor: 3.718

3.  Calcium-dependent enzyme activation and vacuole formation in the apical granular region of pancreatic acinar cells.

Authors:  M Raraty; J Ward; G Erdemli; C Vaillant; J P Neoptolemos; R Sutton; O H Petersen
Journal:  Proc Natl Acad Sci U S A       Date:  2000-11-21       Impact factor: 11.205

4.  Deletion of TRPC3 in mice reduces store-operated Ca2+ influx and the severity of acute pancreatitis.

Authors:  Min Seuk Kim; Jeong Hee Hong; Qin Li; Dong Min Shin; Joel Abramowitz; Lutz Birnbaumer; Shmuel Muallem
Journal:  Gastroenterology       Date:  2009-07-19       Impact factor: 22.682

5.  Caffeine inhibits the agonist-evoked cytosolic Ca2+ signal in mouse pancreatic acinar cells by blocking inositol trisphosphate production.

Authors:  E C Toescu; S C O'Neill; O H Petersen; D A Eisner
Journal:  J Biol Chem       Date:  1992-11-25       Impact factor: 5.157

6.  Acute interstitial pancreatitis in the rat induced by excessive doses of a pancreatic secretagogue.

Authors:  M Lampel; H F Kern
Journal:  Virchows Arch A Pathol Anat Histol       Date:  1977-03-11

7.  Ribosome-free terminals of rough ER allow formation of STIM1 puncta and segregation of STIM1 from IP(3) receptors.

Authors:  Gyorgy Lur; Lee P Haynes; Ian A Prior; Oleg V Gerasimenko; Stefan Feske; Ole H Petersen; Robert D Burgoyne; Alexei V Tepikin
Journal:  Curr Biol       Date:  2009-09-17       Impact factor: 10.834

Review 8.  The role of Ca2+ in the pathophysiology of pancreatitis.

Authors:  Julia V Gerasimenko; Oleg V Gerasimenko; Ole H Petersen
Journal:  J Physiol       Date:  2013-07-29       Impact factor: 5.182

9.  Interactions of antagonists with subtypes of inositol 1,4,5-trisphosphate (IP3) receptor.

Authors:  Huma Saleem; Stephen C Tovey; Tedeusz F Molinski; Colin W Taylor
Journal:  Br J Pharmacol       Date:  2014-07       Impact factor: 8.739

Review 10.  Inositol 1,4,5-trisphosphate receptors and their protein partners as signalling hubs.

Authors:  David L Prole; Colin W Taylor
Journal:  J Physiol       Date:  2016-02-24       Impact factor: 5.182
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  2 in total

1.  Brake adjustment: Ca2+ entry pathway provides a novel target for acute pancreatitis therapy.

Authors:  Richard T Waldron; Aurelia Lugea; Stephen J Pandol
Journal:  Ann Transl Med       Date:  2019-12

2.  Pancreas-specific CHRM3 activation causes pancreatitis in mice.

Authors:  Jianhua Wan; Jiale Wang; Larry E Wagner; Oliver H Wang; Fu Gui; Jiaxiang Chen; Xiaohui Zhu; Ashley N Haddock; Brandy H Edenfield; Brian Haight; Debabrata Mukhopadhyay; Ying Wang; David I Yule; Yan Bi; Baoan Ji
Journal:  JCI Insight       Date:  2021-09-08
  2 in total

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