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7 in total

1. Spontaneous cardiac calcinosis in BALB/cByJ mice.

Authors: Aaron M Glass; Wanda Coombs; Steven M Taffet
Journal: Comp Med Date: 2013-02 Impact factor: 0.982

2. Survey of normal appearing mouse strain which lacks malic enzyme and Nad+-linked glycerol phosphate dehydrogenase: normal pancreatic beta cell function, but abnormal metabolite pattern in skeletal muscle.

Authors: M J MacDonald; L K Marshall
Journal: Mol Cell Biochem Date: 2001-04 Impact factor: 3.396

3. T-cell function is critical for murine cholesterol gallstone formation.

Authors: Kirk J Maurer; Varada P Rao; Zhongming Ge; Arlin B Rogers; Trisha J Oura; Martin C Carey; James G Fox
Journal: Gastroenterology Date: 2007-10 Impact factor: 22.682

4. Anti-tumor necrosis factor VNAR single domains reduce lethality and regulate underlying inflammatory response in a murine model of endotoxic shock.

Authors: Rafael Bojalil; María Teresa Mata-González; Fausto Sánchez-Muñoz; Yepci Yee; Iván Argueta; Lucía Bolaños; Luis Manuel Amezcua-Guerra; Tanya Amanda Camacho-Villegas; Edna Sánchez-Castrejón; Walter Jakob García-Ubbelohde; Alexei Fedorovish Licea-Navarro; Ricardo Márquez-Velasco; Jorge Fernando Paniagua-Solís
Journal: BMC Immunol Date: 2013-04-02 Impact factor: 3.615

Genetic differences in BALB/c sublines.

1. Spontaneous cardiac calcinosis in BALB/cByJ mice.

2. Survey of normal appearing mouse strain which lacks malic enzyme and Nad+-linked glycerol phosphate dehydrogenase: normal pancreatic beta cell function, but abnormal metabolite pattern in skeletal muscle.

3. T-cell function is critical for murine cholesterol gallstone formation.

4. Anti-tumor necrosis factor VNAR single domains reduce lethality and regulate underlying inflammatory response in a murine model of endotoxic shock.

5. An analysis of the role of the indigenous microbiota in cholesterol gallstone pathogenesis.

6. The 5-hydroxytryptamine4 receptor enables differentiation of informational content and encoding in the hippocampus.

7. Comparison of cisplatin-induced anti-tumor response in CT26 syngeneic tumors of three BALB/c substrains.