Literature DB >> 29926469

Benign paroxysmal torticollis, benign paroxysmal vertigo, and benign tonic upward gaze are not benign disorders.

Véronique Humbertclaude1, Benjamin Krams2, Erika Nogue3, Nicolas Nagot3, Daniel Annequin4, Barbara Tourniaire4, Elisabeth Tournier-Lasserve5,6, Florence Riant5,6, Agathe Roubertie2,7.   

Abstract

AIM: Benign paroxysmal torticollis (BPT), benign paroxysmal vertigo (BPV), and benign tonic upward gaze (BTU) are characterized by transient and recurrent episodes of neurological manifestations. The purpose of this study was to analyse the clinical relationships between these syndromes, associated comorbidities, and genetic bases.
METHOD: In this cross-sectional study, clinical data of patients with BPT, BPV, or BTU were collected with a focus on developmental achievements, learning abilities, and rehabilitation. Neuropsychological assessment and genetic testing were performed.
RESULTS: Fifty patients (median age at inclusion 6y) were enrolled. Psychomotor delay, abnormal neurological examination, and low or borderline IQ were found in 19%, 32%, and 26% of the patients respectively. Cognitive dysfunction was present in 27% of the patients. CACNA1A gene mutation was identified in eight families, and KCNA1 and FGF14 mutation in one family respectively. The identification of a CACNA1A mutation was significantly associated with BTU (p=0.03) and with cognitive dysfunction (p=0.01). Patients with BPV were less likely to have cognitive dysfunction.
INTERPRETATION: Children with BPT, BPV, or BTU are at high risk of impaired psychomotor and cognitive development. These syndromes should not be regarded as benign and should be considered as part of the spectrum of a neurodevelopmental disorder. WHAT THIS PAPER ADDS OK: Patients with benign paroxysmal torticollis (BPT), benign paroxysmal vertigo (BPV), and benign tonic upward gaze (BTU) have an increased risk of psychomotor delay. These patients also have an increased risk of abnormal neurological examination and cognitive dysfunction. Gene mutations, especially in CACNA1A, were identified in 21% of the families. BPT, BTU, and BPV should not be regarded as benign. BPT, BTU, and BPV should be considered as part of the spectrum of a neurodevelopmental disorder.
© 2018 Mac Keith Press.

Entities:  

Mesh:

Substances:

Year:  2018        PMID: 29926469     DOI: 10.1111/dmcn.13935

Source DB:  PubMed          Journal:  Dev Med Child Neurol        ISSN: 0012-1622            Impact factor:   5.449


  4 in total

1.  The complexities of CACNA1A in clinical neurogenetics.

Authors:  Marina P Hommersom; Teije H van Prooije; Maartje Pennings; Meyke I Schouten; Hans van Bokhoven; Erik-Jan Kamsteeg; Bart P C van de Warrenburg
Journal:  J Neurol       Date:  2021-11-22       Impact factor: 6.682

Review 2.  Clinical and Genetic Overview of Paroxysmal Movement Disorders and Episodic Ataxias.

Authors:  Giacomo Garone; Alessandro Capuano; Lorena Travaglini; Federica Graziola; Fabrizia Stregapede; Ginevra Zanni; Federico Vigevano; Enrico Bertini; Francesco Nicita
Journal:  Int J Mol Sci       Date:  2020-05-20       Impact factor: 5.923

3.  FGF14-related episodic ataxia: delineating the phenotype of Episodic Ataxia type 9.

Authors:  Julie Piarroux; Florence Riant; Véronique Humbertclaude; Ganaelle Remerand; Jessica Hadjadj; Franck Rejou; Christine Coubes; Lucile Pinson; Pierre Meyer; Agathe Roubertie
Journal:  Ann Clin Transl Neurol       Date:  2020-03-12       Impact factor: 4.511

4.  Benign paroxysmal torticollis: phenotype, natural history, and quality of life.

Authors:  Kaitlin A Greene; Vivien Lu; Marta San Luciano; William Qubty; Samantha L Irwin; Barbara Grimes; Amy A Gelfand
Journal:  Pediatr Res       Date:  2021-02-09       Impact factor: 3.756

  4 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.