Literature DB >> 29909500

High expression of a novel splicing variant of VEGF, L-VEGF144 in glioblastoma multiforme is associated with a poorer prognosis in bevacizumab treatment.

Wen-Yu Cheng1,2, Chiung-Chyi Shen3,4,5,6,7, Ming-Tsang Chiao3, Yea-Jiuan Liang3, Tsuo-Fei Mao7, Bai-Shuan Liu8, Jun-Peng Chen9.   

Abstract

INTRODUCTION: A previous study confirmed that a novel splicing variant of large vascular endothelial growth factor (L-VEGF) termed L-VEGF144, a nucleolus protein, is found in glioblastoma cells and specimens, but the actual biological function and clinical significance of L-VEGF144 remain unclear.
METHODS: In this study, we analyzed the expression of L-VEGF144 in 68 glioblastoma multiforme specimens using reverse transcriptase-polymerase chain reaction analysis.
RESULTS: The results showed that the high expression of L-VEGF144 was associated with a poor prognosis in the bevacizumab plus concurrent chemoradiotherapy with temozolomide treatment. In addition, we constructed a series truncated and mutant form of L-VEGF144 to confirm that exon 6a of L-VEGF144 is able to engage in the nuclear importation and found that 8 lysines within exon 6a play a critical role in the nucleolus aggregation of L-VEGF144. Also, the transfection of the L-VEGF144 increased the number of nucleoli. Furthermore, the recombinant protein Flag-L-VEGF144 and commercial VEGF protein have similar growth stimulatory activities in terms of inducing glioblastoma cell proliferation in vitro.
CONCLUSIONS: Taken together, these results indicated that the expression of L-VEGF144 could potentially serve as an independent indicator of poor prognosis in bevacizumab treatment.

Entities:  

Keywords:  Bevacizumab; GBM Prognosis; Mitogen; Novel VEGF isoform; Nucleolus protein

Mesh:

Substances:

Year:  2018        PMID: 29909500     DOI: 10.1007/s11060-018-2928-z

Source DB:  PubMed          Journal:  J Neurooncol        ISSN: 0167-594X            Impact factor:   4.130


  17 in total

1.  New vascular endothelial growth factor isoform generated by internal ribosome entry site-driven CUG translation initiation.

Authors:  I Huez; S Bornes; D Bresson; L Créancier; H Prats
Journal:  Mol Endocrinol       Date:  2001-12

2.  New isoforms of VEGF are translated from alternative initiation CUG codons located in its 5'UTR.

Authors:  M Meiron; R Anunu; E J Scheinman; S Hashmueli; B Z Levi
Journal:  Biochem Biophys Res Commun       Date:  2001-04-13       Impact factor: 3.575

3.  A precursor form of vascular endothelial growth factor arises by initiation from an upstream in-frame CUG codon.

Authors:  M K Tee; R B Jaffe
Journal:  Biochem J       Date:  2001-10-01       Impact factor: 3.857

4.  Nuclear localization of long-VEGF is associated with hypoxia and tumor angiogenesis.

Authors:  Yifat Rosenbaum-Dekel; Alisa Fuchs; Evgeny Yakirevich; Aviva Azriel; Salam Mazareb; Murray B Resnick; Ben-Zion Levi
Journal:  Biochem Biophys Res Commun       Date:  2005-06-24       Impact factor: 3.575

Review 5.  Role of vascular endothelial growth factor in the regulation of angiogenesis.

Authors:  N Ferrara
Journal:  Kidney Int       Date:  1999-09       Impact factor: 10.612

Review 6.  VEGF Splicing and the Role of VEGF Splice Variants: From Physiological-Pathological Conditions to Specific Pre-mRNA Splicing.

Authors:  Mélanie Guyot; Gilles Pagès
Journal:  Methods Mol Biol       Date:  2015

7.  Suberoylanilide hydroxamic acid (SAHA) causes tumor growth slowdown and triggers autophagy in glioblastoma stem cells.

Authors:  Ming-Tsang Chiao; Wen-Yu Cheng; Yi-Chin Yang; Chiung-Chyi Shen; Jiunn-Liang Ko
Journal:  Autophagy       Date:  2013-08-15       Impact factor: 16.016

Review 8.  Mechanisms of evasive resistance to anti-VEGF therapy in glioblastoma.

Authors:  Kan V Lu; Gabriele Bergers
Journal:  CNS Oncol       Date:  2013-01

9.  Processing of VEGF-A by matrix metalloproteinases regulates bioavailability and vascular patterning in tumors.

Authors:  Sunyoung Lee; Shahla M Jilani; Ganka V Nikolova; Darren Carpizo; M Luisa Iruela-Arispe
Journal:  J Cell Biol       Date:  2005-05-23       Impact factor: 10.539

Review 10.  The 2007 WHO classification of tumours of the central nervous system.

Authors:  David N Louis; Hiroko Ohgaki; Otmar D Wiestler; Webster K Cavenee; Peter C Burger; Anne Jouvet; Bernd W Scheithauer; Paul Kleihues
Journal:  Acta Neuropathol       Date:  2007-07-06       Impact factor: 17.088

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  3 in total

1.  Overexpression of HOXC10 promotes angiogenesis in human glioma via interaction with PRMT5 and upregulation of VEGFA expression.

Authors:  Zhanyao Tan; Kun Chen; Wenjiao Wu; Yanqing Zhou; Jinrong Zhu; Geyan Wu; Lixue Cao; Xin Zhang; Hongyu Guan; Yi Yang; Wei Zhang; Jun Li
Journal:  Theranostics       Date:  2018-10-06       Impact factor: 11.556

2.  A Novel Splice Variant of BCAS1 Inhibits β-Arrestin 2 to Promote the Proliferation and Migration of Glioblastoma Cells, and This Effect Was Blocked by Maackiain.

Authors:  Yun-Hua Kuo; Huey-Shan Hung; Chia-Wen Tsai; Shao-Chih Chiu; Shih-Ping Liu; Yu-Ting Chiang; Woei-Cherng Shyu; Shinn-Zong Lin; Ru-Huei Fu
Journal:  Cancers (Basel)       Date:  2022-08-11       Impact factor: 6.575

Review 3.  Splicing Dysregulation as Oncogenic Driver and Passenger Factor in Brain Tumors.

Authors:  Pamela Bielli; Vittoria Pagliarini; Marco Pieraccioli; Cinzia Caggiano; Claudio Sette
Journal:  Cells       Date:  2019-12-18       Impact factor: 6.600

  3 in total

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