Literature DB >> 29884290

Cardioprotective effect of substance P in a porcine model of acute myocardial infarction.

Doo Sun Sim1, Weon Kim2, Kyung Hye Lee3, Ho Chun Song4, Ja Hye Kim4, Dae Sung Park1, Kyung Seob Lim5, Jong Shin Woo3, Young Joon Hong1, Youngkeun Ahn1, Hyun Sook Hong6, Youngsook Son6, Myung Ho Jeong7.   

Abstract

BACKGROUND: Substance P (SP) may attenuate ischemia-reperfusion injury by reducing inflammation. We assessed cardioprotective effect of SP in a porcine model of acute myocardial infarction (AMI).
METHODS: AMI was induced by occlusion of the left anterior descending artery on 28 swine, randomized to SP 5 nmol/kg (group 1, n = 14) and normal saline (group 2, n = 14) given intravenously 5 min before reperfusion. Blood samples were collected at baseline, 3 days and 4 weeks. Echocardiography and myocardial perfusion single photon emission computed tomography (SPECT) were performed at 1 week and 4 weeks. Histomorphometric infarct size assessment was done at 4 weeks.
RESULTS: Left ventricular (LV) ejection fraction (EF) (LVEF) after AMI induction was higher in group 1 than group 2 (37.9 ± 4.6% vs. 29.4 ± 3.2%, p = 0.001) but not different at 4 weeks. No significant difference was observed in perfusion defect extent and total perfusion defect on SPECT at 1 week and 4 weeks. Pathologic infarct size (% LV) was significantly smaller in group 1 than group 2 (2.4 ± 2.3% vs. 5.7 ± 2.5%, p = 0.020). The ratio of neutrophil to lymphocyte on day 3 and serum creatinine concentration at 4 weeks after AMI were lower in group 1.
CONCLUSIONS: In a porcine model of AMI, SP improved LVEF early post-MI and reduced infarct size. SP may be beneficial in reducing inflammation and ischemia-reperfusion injury after AMI.
Copyright © 2018 Elsevier B.V. All rights reserved.

Entities:  

Keywords:  Myocardial infarction; Reperfusion; Ventricular remodeling

Mesh:

Substances:

Year:  2018        PMID: 29884290     DOI: 10.1016/j.ijcard.2018.05.113

Source DB:  PubMed          Journal:  Int J Cardiol        ISSN: 0167-5273            Impact factor:   4.164


  3 in total

1.  Substance P Administered after Myocardial Infarction Upregulates Microphthalmia-Associated Transcription Factor, GATA4, and the Expansion of c-Kit+ Cells.

Authors:  Yun-Mi Jeong; Xian Wu Cheng; Weon Kim
Journal:  Stem Cells Int       Date:  2020-02-10       Impact factor: 5.443

2.  Replacement of Lost Substance P Reduces Fibrosis in the Diabetic Heart by Preventing Adverse Fibroblast and Macrophage Phenotype Changes.

Authors:  Alexander Widiapradja; Ainsley O Kasparian; Samuel L McCaffrey; Lauren L Kolb; John D Imig; Jessica L Lacey; Giselle C Melendez; Scott P Levick
Journal:  Cells       Date:  2021-10-05       Impact factor: 6.600

3.  Substance P enhances the local activation of NK1R-expressing c-kit+ cardiac progenitor cells in right atrium of ischemia/reperfusion-injured heart.

Authors:  Yun-Mi Jeong; Xian Wu Cheng; Kyung Hye Lee; Sora Lee; Haneul Cho; Weon Kim
Journal:  BMC Mol Cell Biol       Date:  2020-06-09
  3 in total

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