Literature DB >> 29874175

ExomeChip-Wide Analysis of 95 626 Individuals Identifies 10 Novel Loci Associated With QT and JT Intervals.

Nathan A Bihlmeyer1, Jennifer A Brody1, Albert Vernon Smith1, Helen R Warren1, Honghuang Lin1, Aaron Isaacs1, Ching-Ti Liu1, Jonathan Marten1, Farid Radmanesh1, Leanne M Hall1, Niels Grarup1, Hao Mei1, Martina Müller-Nurasyid1, Jennifer E Huffman1, Niek Verweij1, Xiuqing Guo1, Jie Yao1, Ruifang Li-Gao1, Marten van den Berg1, Stefan Weiss1, Bram P Prins1, Jessica van Setten1, Jeffrey Haessler1, Leo-Pekka Lyytikäinen1, Man Li1, Alvaro Alonso1, Elsayed Z Soliman1, Joshua C Bis1, Tom Austin1, Yii-Der Ida Chen1, Bruce M Psaty1, Tamara B Harrris1, Lenore J Launer1, Sandosh Padmanabhan1, Anna Dominiczak1, Paul L Huang1, Zhijun Xie1, Patrick T Ellinor1, Jan A Kors1, Archie Campbell1, Alison D Murray1, Christopher P Nelson1, Martin D Tobin1, Jette Bork-Jensen1, Torben Hansen1, Oluf Pedersen1, Allan Linneberg1, Moritz F Sinner1, Annette Peters1, Melanie Waldenberger1, Thomas Meitinger1, Siegfried Perz1, Ivana Kolcic1, Igor Rudan1, Rudolf A de Boer1, Peter van der Meer1, Henry J Lin1, Kent D Taylor1, Renée de Mutsert1, Stella Trompet1, J Wouter Jukema1, Arie C Maan1, Bruno H C Stricker1, Fernando Rivadeneira1, André Uitterlinden1, Uwe Völker1, Georg Homuth1, Henry Völzke1, Stephan B Felix1, Massimo Mangino1, Timothy D Spector1, Michiel L Bots1, Marco Perez1, Olli T Raitakari1, Mika Kähönen1, Nina Mononen1, Vilmundur Gudnason1, Patricia B Munroe1, Steven A Lubitz1, Cornelia M van Duijn1, Christopher H Newton-Cheh1, Caroline Hayward1, Jonathan Rosand1, Nilesh J Samani1, Jørgen K Kanters1, James G Wilson1, Stefan Kääb1, Ozren Polasek1, Pim van der Harst1, Susan R Heckbert1, Jerome I Rotter1, Dennis O Mook-Kanamori1, Mark Eijgelsheim1, Marcus Dörr1, Yalda Jamshidi1, Folkert W Asselbergs1, Charles Kooperberg1, Terho Lehtimäki1, Dan E Arking1, Nona Sotoodehnia1.   

Abstract

BACKGROUND: QT interval, measured through a standard ECG, captures the time it takes for the cardiac ventricles to depolarize and repolarize. JT interval is the component of the QT interval that reflects ventricular repolarization alone. Prolonged QT interval has been linked to higher risk of sudden cardiac arrest. METHODS AND
RESULTS: We performed an ExomeChip-wide analysis for both QT and JT intervals, including 209 449 variants, both common and rare, in 17 341 genes from the Illumina Infinium HumanExome BeadChip. We identified 10 loci that modulate QT and JT interval duration that have not been previously reported in the literature using single-variant statistical models in a meta-analysis of 95 626 individuals from 23 cohorts (comprised 83 884 European ancestry individuals, 9610 blacks, 1382 Hispanics, and 750 Asians). This brings the total number of ventricular repolarization associated loci to 45. In addition, our approach of using coding variants has highlighted the role of 17 specific genes for involvement in ventricular repolarization, 7 of which are in novel loci.
CONCLUSIONS: Our analyses show a role for myocyte internal structure and interconnections in modulating QT interval duration, adding to previous known roles of potassium, sodium, and calcium ion regulation, as well as autonomic control. We anticipate that these discoveries will open new paths to the goal of making novel remedies for the prevention of lethal ventricular arrhythmias and sudden cardiac arrest.
© 2018 The Authors.

Entities:  

Keywords:  arrhythmias, cardiac; death, sudden, cardiac; genetics; genome; humans

Mesh:

Substances:

Year:  2018        PMID: 29874175      PMCID: PMC5992491          DOI: 10.1161/CIRCGEN.117.001758

Source DB:  PubMed          Journal:  Circ Genom Precis Med        ISSN: 2574-8300


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