Literature DB >> 29868839

An in silico proteomics screen to predict and prioritize protein-protein interactions dependent on post-translationally modified motifs.

Anna M Schmoker1, Heather E Driscoll2,3, Stefanie R Geiger1, James J Vincent1,2, Alicia M Ebert1, Bryan A Ballif1.   

Abstract

Motivation: The development of proteomic methods for the characterization of domain/motif interactions has greatly expanded our understanding of signal transduction. However, proteomics-based binding screens have limitations including that the queried tissue or cell type may not harbor all potential interacting partners or post-translational modifications (PTMs) required for the interaction. Therefore, we sought a generalizable, complementary in silico approach to identify potentially novel motif and PTM-dependent binding partners of high priority.
Results: We used as an initial example the interaction between the Src homology 2 (SH2) domains of the adaptor proteins CT10 regulator of kinase (CRK) and CRK-like (CRKL) and phosphorylated-YXXP motifs. Employing well-curated, publicly-available resources, we scored and prioritized potential CRK/CRKL-SH2 interactors possessing signature characteristics of known interacting partners. Our approach gave high priority scores to 102 of the >9000 YXXP motif-containing proteins. Within this 102 were 21 of the 25 curated CRK/CRKL-SH2-binding partners showing a more than 80-fold enrichment. Several predicted interactors were validated biochemically. To demonstrate generalized applicability, we used our workflow to predict protein-protein interactions dependent upon motif-specific arginine methylation. Our data demonstrate the applicability of our approach to, conceivably, any modular binding domain that recognizes a specific post-translationally modified motif. Supplementary information: Supplementary data are available at Bioinformatics online.

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Year:  2018        PMID: 29868839      PMCID: PMC6223376          DOI: 10.1093/bioinformatics/bty434

Source DB:  PubMed          Journal:  Bioinformatics        ISSN: 1367-4803            Impact factor:   6.937


  46 in total

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Authors:  David Chodniewicz; Richard L Klemke
Journal:  Biochim Biophys Acta       Date:  2004-07-05

Review 2.  Reading protein modifications with interaction domains.

Authors:  Bruce T Seet; Ivan Dikic; Ming-Ming Zhou; Tony Pawson
Journal:  Nat Rev Mol Cell Biol       Date:  2006-07       Impact factor: 94.444

3.  The noncatalytic src homology region 2 segment of abl tyrosine kinase binds to tyrosine-phosphorylated cellular proteins with high affinity.

Authors:  B J Mayer; P K Jackson; D Baltimore
Journal:  Proc Natl Acad Sci U S A       Date:  1991-01-15       Impact factor: 11.205

Review 4.  Arginine Methylation: The Coming of Age.

Authors:  Roméo S Blanc; Stéphane Richard
Journal:  Mol Cell       Date:  2017-01-05       Impact factor: 17.970

5.  Defining Human Tyrosine Kinase Phosphorylation Networks Using Yeast as an In Vivo Model Substrate.

Authors:  Thomas Corwin; Jonathan Woodsmith; Federico Apelt; Jean-Fred Fontaine; David Meierhofer; Johannes Helmuth; Arndt Grossmann; Miguel A Andrade-Navarro; Bryan A Ballif; Ulrich Stelzl
Journal:  Cell Syst       Date:  2017-08-23       Impact factor: 10.304

6.  SMN, the product of the spinal muscular atrophy gene, binds preferentially to dimethylarginine-containing protein targets.

Authors:  W J Friesen; S Massenet; S Paushkin; A Wyce; G Dreyfuss
Journal:  Mol Cell       Date:  2001-05       Impact factor: 17.970

7.  Crk and Crk-like play essential overlapping roles downstream of disabled-1 in the Reelin pathway.

Authors:  Tae-Ju Park; Tom Curran
Journal:  J Neurosci       Date:  2008-12-10       Impact factor: 6.167

8.  Determinants of the interaction of the spinal muscular atrophy disease protein SMN with the dimethylarginine-modified box H/ACA small nucleolar ribonucleoprotein GAR1.

Authors:  Sarah E Whitehead; Kevin W Jones; Xing Zhang; Xiaodong Cheng; Rebecca M Terns; Michael P Terns
Journal:  J Biol Chem       Date:  2002-09-19       Impact factor: 5.157

9.  Diverse spermatogenic defects in humans caused by Y chromosome deletions encompassing a novel RNA-binding protein gene.

Authors:  R Reijo; T Y Lee; P Salo; R Alagappan; L G Brown; M Rosenberg; S Rozen; T Jaffe; D Straus; O Hovatta
Journal:  Nat Genet       Date:  1995-08       Impact factor: 38.330

10.  CAS/Crk coupling serves as a "molecular switch" for induction of cell migration.

Authors:  R L Klemke; J Leng; R Molander; P C Brooks; K Vuori; D A Cheresh
Journal:  J Cell Biol       Date:  1998-02-23       Impact factor: 10.539

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