Literature DB >> 29768263

Fibrinopeptide A Induces Expression of C-Reactive Protein via the ROS-ERK1/2/ P38-NF-κB Signal Pathway in Vascular Smooth Muscle Cells.

Shouzhu Xu1, Jing Zhao1, Juntian Liu1,2, Wei Gou1.   

Abstract

BACKGROUND/AIMS: Atherosclerosis is a chronic inflammatory disease in the artery walls. Fibrinopeptide A (FPA) is a biomarker of the activation of coagulation system, and a high concentration of FPA in blood occurs in patients with ischemic heart disease etc. However, there exist few studies on the pathological effects of FPA in cardiovascular system. Therefore, the present study examined the effect of FPA on CRP expression in VSMCs and the molecular mechanisms.
METHODS: mRNA and protein expression was identified by quantitative real-time PCR and Western blot, respectively. Reactive oxygen species (ROS) and the immunofluorescence staining were observed by a fluorescence microscope. Plasma FPA and CRP level was determined by ELISA.
RESULTS: FPA induced the expressions of CRP, IL-1β and IL-6 in VSMCs, and anti-IL-1β and anti-IL-6 neutralizing antibodies partially reduced FPA-induced CRP expression in VSMCs. The subchronic administration of FPA to rats increased FPA level in plasma and CRP expression in the aortic artery walls. The further studies showed that FPA promoted superoxide anion generation in VSMCs. Antioxidant NAC antagonized FPA-stimulated superoxide anion generation and inhibited FPA-induced CRP expression in VSMCs. FPA activated ERK1/2 and p38 phosphorylation, and PD98059 and SB203580 reduced FPA-induced CRP expression. Moreover, NAC inhibited the activation of ERK1/2 and p38. In addition, FPA enhanced NF-κB level in the nuclei of VSMCs, and PDTC reduced FPA-induced expression of CRP.
CONCLUSIONS: FPA induces CRP expression in VSMCs via ROS-ERK1/2/p38-NF-κB signal pathway. This finding for the first time provides an experimental evidence for pro-inflammatory effect of FPA.
© 2018 The Author(s). Published by S. Karger AG, Basel.

Entities:  

Keywords:  Atherosclerosis; CRP; Fibrinopeptide A; Inflammation; Vascular smooth muscle cells

Mesh:

Substances:

Year:  2018        PMID: 29768263     DOI: 10.1159/000489805

Source DB:  PubMed          Journal:  Cell Physiol Biochem        ISSN: 1015-8987


  5 in total

Review 1.  Patient Stratification in Sepsis: Using Metabolomics to Detect Clinical Phenotypes, Sub-Phenotypes and Therapeutic Response.

Authors:  Humma Hussain; Kritchai Vutipongsatorn; Beatriz Jiménez; David B Antcliffe
Journal:  Metabolites       Date:  2022-04-21

2.  Inhibitory Effect of Curcumin on Artery Restenosis Following Carotid Endarterectomy and Its Associated Mechanism in vitro and in vivo.

Authors:  Dapeng Zhang; Yanhui Yang; Yuanchao Li; Guodong Zhang; Zhenguo Cheng
Journal:  Drug Des Devel Ther       Date:  2020-02-26       Impact factor: 4.162

3.  Elucidating the endogenous synovial fluid proteome and peptidome of inflammatory arthritis using label-free mass spectrometry.

Authors:  Shalini M Mahendran; Edward C Keystone; Roman J Krawetz; Kun Liang; Eleftherios P Diamandis; Vinod Chandran
Journal:  Clin Proteomics       Date:  2019-05-30       Impact factor: 3.988

4.  Effects of Hyperbaric Oxygen on T helper 17/regulatory T Polarization in Antigen and Collagen-induced Arthritis: Hypoxia-inducible Factor-1α as a Target.

Authors:  Titut Harnanik; Joewono Soeroso; Mohammad Guritno Suryokusumo; Tedy Juliandhy
Journal:  Oman Med J       Date:  2020-01-23

5.  Impact of C-reactive protein on osteo-/chondrogenic transdifferentiation and calcification of vascular smooth muscle cells.

Authors:  Laura A Henze; Trang T D Luong; Beate Boehme; Jaber Masyout; Markus P Schneider; Sebastian Brachs; Florian Lang; Burkert Pieske; Andreas Pasch; Kai-Uwe Eckardt; Jakob Voelkl; Ioana Alesutan
Journal:  Aging (Albany NY)       Date:  2019-08-03       Impact factor: 5.682

  5 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.