Literature DB >> 29759572

Early Arrhythmic Events in Idiopathic Dilated Cardiomyopathy.

Pasquale Losurdo1, Davide Stolfo2, Marco Merlo2, Giulia Barbati2, Marco Gobbo2, Marta Gigli2, Federica Ramani2, Bruno Pinamonti2, Massimo Zecchin2, Gherardo Finocchiaro3, Luisa Mestroni4, Gianfranco Sinagra2.   

Abstract

OBJECTIVES: The study sought to provide an insight into the prevalence, characterization and possible reliable indicators of early sudden cardiac death/malignant ventricular arrhythmias (SCD/MVAs) in a large cohort of dilated cardiomyopathy (DCM).
BACKGROUND: DCM generally affects young individuals and is characterized by an unpredictable prognosis with a non-negligible risk of SCD/MVAs, particularly in early stages of disease.
METHODS: From 1988 to 2014, 952 patients with DCM were consecutively included in the Heart Muscle Disease Registry of Trieste.
RESULTS: Globally, 20 patients (2.1% of the overall population) experienced SCD/MVAs within the first 6 months after enrollment (primary endpoint). At baseline they showed a worse functional class (New York Heart Association functional class III to IV 42% vs. 22%, p = 0.038), a longer QRS complex duration (127 ± 41 ms vs. 108 ± 33 ms; p = 0.013) and a larger indexed left ventricular end-systolic volume (LVESVI) (82 ± 49 ml/m2 vs. 67 ± 34 ml/m2; p = 0.049), as compared to patients without early SCD/MVAs. Beta-blockers were less tolerated (59% vs. 83% in patients with no early SCD/MVAs; p = 0.008), mostly due to hemodynamic intolerance. At multivariate analysis, LVESVI (odds ratio [OR]: 1.012; 95% confidence interval [CI]: 1.000 to 1.024; p = 0.043) and QRS complex duration (OR: 1.017; 95% CI: 1.003 to 1.030; p = 0.015) were independently associated with the primary endpoint, whereas beta-blockers demonstrated a protective effect (OR: 0.169, CI: 0.048 to 0.593; p = 0.006).
CONCLUSIONS: Patients with DCM present a significant risk of major arrhythmic events in the first phase of the disease. Baseline LVESVI, QRS duration, and intolerance to beta-blocker therapy might be useful tools in the arrhythmic early risk assessment.
Copyright © 2016 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved.

Entities:  

Keywords:  arrhythmia; cardiomyopathy; sudden death

Year:  2016        PMID: 29759572     DOI: 10.1016/j.jacep.2016.05.002

Source DB:  PubMed          Journal:  JACC Clin Electrophysiol        ISSN: 2405-500X


  4 in total

1.  Transcriptome signature of ventricular arrhythmia in dilated cardiomyopathy reveals increased fibrosis and activated TP53.

Authors:  Mary E Haywood; Andrea Cocciolo; Kadijah F Porter; Evgenia Dobrinskikh; Dobromir Slavov; Sharon L Graw; T Brett Reece; Amrut V Ambardekar; Michael R Bristow; Luisa Mestroni; Matthew R G Taylor
Journal:  J Mol Cell Cardiol       Date:  2020-01-18       Impact factor: 5.000

Review 2.  Ventricular arrhythmias and ARNI: is it time to reappraise their management in the light of new evidence?

Authors:  Andrea Lorenzo Vecchi; Raffaele Abete; Jacopo Marazzato; Attilio Iacovoni; Andrea Mortara; Roberto De Ponti; Michele Senni
Journal:  Heart Fail Rev       Date:  2022-01       Impact factor: 4.214

Review 3.  Reverse remodeling in Dilated Cardiomyopathy: Insights and future perspectives.

Authors:  M Merlo; T Caiffa; M Gobbo; L Adamo; G Sinagra
Journal:  Int J Cardiol Heart Vasc       Date:  2018-03-08

Review 4.  Dilated cardiomyopathy in the era of precision medicine: latest concepts and developments.

Authors:  Nicoletta Orphanou; Efstathios Papatheodorou; Aris Anastasakis
Journal:  Heart Fail Rev       Date:  2021-07-14       Impact factor: 4.654

  4 in total

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